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MAB1628

Sigma-Aldrich

抗肌球蛋白抗体(慢肌,克隆NOQ7.5.4D)

clone NOQ7.5.4D, Chemicon®, from mouse

别名:

Anti-CMD1S, Anti-CMH1, Anti-MPD1, Anti-MYHCB, Anti-SPMD, Anti-SPMM

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About This Item

UNSPSC代码:
12352203
eCl@ss:
32160702
NACRES:
NA.41

生物来源

mouse

质量水平

抗体形式

purified immunoglobulin

抗体产品类型

primary antibodies

克隆

NOQ7.5.4D, monoclonal

种属反应性

rat, feline, human

制造商/商品名称

Chemicon®

技术

immunohistochemistry: suitable
radioimmunoassay: suitable
western blot: suitable

同位素/亚型

IgG

NCBI登记号

UniProt登记号

运输

wet ice

靶向翻译后修饰

unmodified

基因信息

human ... MYH7B(57644)

特异性

慢型肌球蛋白重链明确识别1型纤维。在骨骼肌中,MAB1628 对于各种物种的慢型肌球蛋白重链有特异性。它与大鼠和猫慢型肌球蛋白重链发生强烈反应。MAB1628 也识别心室中的β(慢型)肌球蛋白重链。

免疫原

从组织化学法混合的人骨骼肌分离的肌原纤维中纯化的肌球蛋白。
表位:慢肌

应用

免疫组化:冰冻和福尔马林固定切片。

免疫印迹

RIA

最佳工作稀释度必须由最终用户确定。
抗肌球蛋白抗体(慢肌,克隆NOQ7.5.4D)是针对肌球蛋白的抗体,用于RIA,、WB、IH。

外形

形式:纯化

其他说明

浓度:请参考批次特异性浓缩物的检验报告。

法律信息

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany

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WGK

WGK 2

闪点(°F)

Not applicable

闪点(°C)

Not applicable


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Linda L Kusner et al.
Investigative ophthalmology & visual science, 51(1), 192-200 (2009-08-08)
Extraocular muscle (EOM) has a distinct skeletal muscle phenotype. The hypothesis for the study was that fibroblasts support the unique EOM phenotype and that perimysial fibroblasts derived from EOM have properties that distinguish them from fibroblasts derived from other skeletal
Camila Silva Foresto et al.
Journal of applied physiology (Bethesda, Md. : 1985), 121(3), 646-660 (2016-07-23)
Muscle loss occurs following injury and immobilization in adulthood and childhood, which impairs the rehabilitation process; however, far fewer studies have been conducted analyzing atrophic response in infants. This work investigated first the morphological and molecular mechanisms involved in immobilization-induced
Lipid in skeletal muscle myotubes is associated to the donors' insulin sensitivity and physical activity phenotypes.
Bajpeyi, S; Myrland, CK; Covington, JD; Obanda, D; Cefalu, WT; Smith, SR; Rustan, AC; Ravussin, E
Obesity (Silver Spring, Md.) null
Jenny Lund et al.
Scientific reports, 8(1), 9814-9814 (2018-07-01)
Once assumed only to be a waste product of anaerobe glycolytic activity, lactate is now recognized as an energy source in skeletal muscles. While lactate metabolism has been extensively studied in vivo, underlying cellular processes are poorly described. This study
Christian M Girgis et al.
Journal of cachexia, sarcopenia and muscle, 10(6), 1228-1240 (2019-06-22)
It has long been recognized that vitamin D deficiency is associated with muscle weakness and falls. Vitamin D receptor (VDR) is present at very low levels in normal muscle. Whether vitamin D plays a direct role in muscle function is

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