产品名称
大鼠/小鼠胰岛素ELISA, This Rat/Mouse Insulin ELISA is used to measure & quantify Insulin levels in Metabolism & Endocrine research.
species reactivity
mouse, rat
packaging
kit of 1 × 96 wells
parameter
10 μL sample volume (4hr assay)
assay range
accuracy: 83-102%
linearity: 79-133%
sensitivity: 0.2 ng/mL
standard curve range: 0.2-10 ng/mL
technique(s)
ELISA: suitable
input
sample type serum
sample type plasma (K2 EDTA)
application(s)
research use
detection method
colorimetric (450nm/590nm)
shipped in
wet ice
storage temp.
2-8°C
Quality Level
Application
大鼠/小鼠胰岛素ELISA适用于代谢和内分泌研究中胰岛素水平的测定和量化。它已被用于通过酶联免疫吸附分析测定血浆胰岛素。它也被用于测定细胞的体外胰岛素分泌。
Biochem/physiol Actions
胰岛素是维持正常血液葡萄糖水平的关键激素。它介导细胞葡萄糖摄取，控制碳水化合物、脂质和葡萄糖代谢，并通过有丝分裂效应促进细胞分裂和生长。胰岛素抵抗是对胰岛素介导的葡萄糖处理的敏感度下降的病症。
Disclaimer
仅供研究使用。不用于诊断操作。
General description
胰岛素由胰腺的胰岛细胞分泌。它包含两条由二硫键连接的肽链。A链由21个氨基酸组成，B链具有30个氨基酸。
Other Notes
3 小时测定，室温 10 μL 样品量
研究类别：代谢
研究子类别：肥胖/代谢紊乱
研究类别：代谢
研究子类别：肥胖/代谢紊乱
signalword
Warning
hcodes
Hazard Classifications
Met. Corr. 1 - Skin Sens. 1
存储类别
8A - Combustible corrosive hazardous materials
Hypoxia-induced intrauterine growth restriction increases the susceptibility of rats to high-fat diet-induced metabolic syndrome.
Rueda-Clausen CF, Dolinsky VW, Morton JS, Proctor SD, Dyck JR, Davidge ST.
Diabetes null
Offspring metabolomic response to maternal protein restriction in a rat model of intrauterine growth restriction (IUGR).
Alexandre-Gouabau, Marie-Cecile, et al.
Journal of Proteome Research, 10, 3292-3302 (2011)
Amadori-glycated phosphatidylethanolamine, a potential marker for hyperglycemia, in streptozotocin-induced diabetic rats.
Sookwong P, Nakagawa K, Fujita I, Shoji N, Miyazawa T.
Lipids null
Chronic hypoxia increases insulin-stimulated glucose uptake in mouse soleus muscle.
Gamboa JL, Garcia-Cazarin ML, Andrade FH
American Journal of Physiology. Regulatory, Integrative and Comparative Physiology null
Interactions Between Age, Stress and Insulin on Cognition: Implications for Alzheimer's Disease.
Solas M, Aisa B, Mugueta MC, Del R?-o J, Tordera RM, Ram?-rez MJ
Neuropsychopharmacology null
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