推荐产品
生物来源
goat
质量水平
抗体形式
serum
抗体产品类型
primary antibodies
克隆
polyclonal
种属反应性
human
制造商/商品名称
Chemicon®
技术
immunoprecipitation (IP): suitable
western blot: suitable
NCBI登记号
UniProt登记号
运输
dry ice
靶向翻译后修饰
unmodified
基因信息
human ... APOB(338)
特异性
载脂蛋白B.单特异性，采用纯抗体与2X合并人血清和纯合并人血浆进行IEP检测。
免疫原
纯化的人载脂蛋白B。
应用
抗载脂蛋白B抗体是一种用于IP &WB的抗载脂蛋白B抗体。
研究子类别
脂代谢 & 体重调节
脂代谢 & 体重调节
研究类别
代谢
代谢
蛋白质印迹
免疫沉淀
最佳工作稀释度必须由最终用户确定。
免疫沉淀
最佳工作稀释度必须由最终用户确定。
外形
根据需要通过固相色谱法进行去纤维蛋白、脱脂和吸收。 在0.05 M Tris-HCl pH 7.5、0.5 M NaCl和0.1%叠氮化钠中的山羊抗血清。
储存及稳定性
以未稀释的等分试样保存于-20°C下12个月。应避免反复冻/融循环。
分析说明
对照
肝脏，HepG2细胞
肝脏，HepG2细胞
法律信息
CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany
免责声明
除非我们的产品目录或产品附带的其他公司文档另有说明，否则我们的产品仅供研究使用，不得用于任何其他目的，包括但不限于未经授权的商业用途、体外诊断用途、离体或体内治疗用途或任何类型的消费或应用于人类或动物。
WGK
WGK 1
Lee G D Fryer et al.
The Journal of biological chemistry, 289(7), 4244-4261 (2013-12-18)
Triglycerides and cholesterol are essential for life in most organisms. Triglycerides serve as the principal energy storage depot and, where vascular systems exist, as a means of energy transport. Cholesterol is essential for the functional integrity of all cellular membrane
Jelske N van der Veen et al.
Journal of lipid research, 58(4), 656-667 (2017-02-06)
Mice lacking phosphatidylethanolamine N-methyltransferase (PEMT) are protected from high-fat diet (HFD)-induced obesity and insulin resistance. However, these mice develop severe nonalcoholic fatty liver disease (NAFLD) when fed the HFD, which is mainly due to inadequate secretion of VLDL particles. Our
Ubiquitination regulates the assembly of VLDL in HepG2 cells and is the committing step of the apoB-100 ERAD pathway.
Fisher, EA; Khanna, NA; McLeod, RS
Journal of Lipid Research null
Erythrocyte-bound apolipoprotein B in relation to atherosclerosis, serum lipids and ABO blood group.
Klop, B; van de Geijn, GJ; Bovenberg, SA; van der Meulen, N; Elte, JW; Birnie, E; Njo et al.
Testing null
Rene C Adam et al.
Journal of lipid research, 61(9), 1271-1286 (2020-07-11)
Angiopoietin-like protein (ANGPTL)3 regulates plasma lipids by inhibiting LPL and endothelial lipase (EL). ANGPTL3 inactivation lowers LDL-C independently of the classical LDLR-mediated pathway and represents a promising therapeutic approach for individuals with homozygous familial hypercholesterolemia due to LDLR mutations. Yet
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