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Merck
CN

657012

兔抗酪氨酸羟化酶多克隆抗体

liquid, Calbiochem®

别名:

抗酪氨酸羟化酶抗体

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关于此项目

NACRES:
NA.41
UNSPSC Code:
12352203
Clone:
polyclonal
Species reactivity:
-
Application:
Citations:
87
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biological source

rabbit

Quality Level

antibody form

purified antibody

antibody product type

primary antibodies

clone

polyclonal

form

liquid

does not contain

preservative

species reactivity (predicted by homology)

all

manufacturer/tradename

Calbiochem®

storage condition

OK to freeze, avoid repeated freeze/thaw cycles

dilution

(Dot Blot (1:1000)
Immunoblotting (1:1000)
Immunofluorescence (1:1000)
Frozen Sections (1:1000; )

isotype

IgG

shipped in

wet ice

storage temp.

−70°C

target post-translational modification

unmodified

General description

亲和纯化的兔多克隆抗体。识别约60 kDa的酪氨酸羟化酶蛋白。
在识别冈田酸处理的PC12细胞中约60 kDa的酪氨酸羟化酶蛋白。
该抗酪氨酸羟化酶兔pAb经验证可用于斑点印迹、免疫印迹、免疫荧光、冷冻切片检测酪氨酸羟化酶

Immunogen

大鼠
纯化SDS变性大鼠嗜铬细胞瘤酪氨酸羟化酶

Application

斑点杂交(1:1000)

免疫印迹(1:1000)

免疫荧光(1:1000)

冰冻切片(1:1000;见应用参考)

Physical form

在150 mM NaCl、10 mM HEPES、50%丙三醇、0.01% BSA、pH 7.5中。

Preparation Note

初次解冻后,分装冻存(-70°C)。

Legal Information

CALBIOCHEM is a registered trademark of Merck KGaA, Darmstadt, Germany

Disclaimer

毒性:标准处理(A)


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存储类别

10 - Combustible liquids

wgk

WGK 1



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Kanako Otomo et al.
Nature communications, 11(1), 6286-6286 (2020-12-10)
The in vivo firing patterns of ventral midbrain dopamine neurons are controlled by afferent and intrinsic activity to generate sensory cue and prediction error signals that are essential for reward-based learning. Given the absence of in vivo intracellular recordings during
A K M Ghulam Muhammad et al.
Clinical cancer research : an official journal of the American Association for Cancer Research, 15(19), 6113-6127 (2009-10-01)
Glioblastoma multiforme is a deadly primary brain cancer. Because the tumor kills due to recurrences, we tested the hypothesis that a new treatment would lead to immunological memory in a rat model of recurrent glioblastoma multiforme. We developed a combined
Ximena I Salinas-Hernández et al.
eLife, 7 (2018-11-14)
Extinction of fear responses is critical for adaptive behavior and deficits in this form of safety learning are hallmark of anxiety disorders. However, the neuronal mechanisms that initiate extinction learning are largely unknown. Here we show, using single-unit electrophysiology and



全球贸易项目编号

货号GTIN
657012-100UL04055977184778