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一般描述
Goat polyclonal antibody supplied as undiluted serum. Recognizes Factor B.
Recognizes Factor B.
This Anti-Factor B Goat pAb is validated for use in Immunoelectrophoresis, Radial Immunodiffusion, Ouchterlony for the detection of Factor B.
免疫原
Human
purified, human factor B
应用
Immunoelectrophoresis (see comments)
Radial Immunodiffusion (see comments)
Ouchterlony (≥ undiluted)
Radial Immunodiffusion (see comments)
Ouchterlony (≥ undiluted)
包装
Please refer to vial label for lot-specific concentration.
警告
Toxicity: Standard Handling (A)
重悬
Following initial thaw, aliquot and freeze (-20°C).
其他说明
This antibody has also been reported to work for immunoelectrophoresis and radial immunodiffusion. Variables associated with assay conditions will dictate the proper working dilution.
法律信息
CALBIOCHEM is a registered trademark of Merck KGaA, Darmstadt, Germany
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储存分类代码
10 - Combustible liquids
WGK
WGK 1
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Prolonged ischemia of skeletal muscle tissue, followed by reperfusion, leads to ischemia/reperfusion injury (IRI), which is a feared local and systemic inflammatory reaction. With respect to the 3Rs, we wanted to determine which parameters for assessment of IRI require a
Journal of clinical immunology, 36(5), 517-527 (2016-05-06)
Autoantibodies termed C3-nephritic factor (C3NeF), which stabilize convertases of the alternative complement pathway, often stimulate autoinflammatory diseases. However, knowledge about analogous autoantibodies acting on the classical pathway (C4NeF) is limited to a few reports, which indicate association with kidney dysfunction
Cell reports, 29(9), 2835-2848 (2019-11-28)
Complement dysregulation is a feature of many retinal diseases, yet mechanistic understanding at the cellular level is limited. Given this knowledge gap about which retinal cells express complement, we performed single-cell RNA sequencing on ∼92,000 mouse retinal cells and validated
PloS one, 8(8), e72059-e72059 (2013-08-31)
Ischemia/reperfusion injury of lower extremities and associated lung damage may result from thrombotic occlusion, embolism, trauma, or surgical intervention with prolonged ischemia and subsequent restoration of blood flow. This clinical entity is characterized by high morbidity and mortality. Deprivation of
Frontiers in immunology, 15, 1334151-1334151 (2024-06-26)
Complement factor H (FH) is a major regulator of the complement alternative pathway, its mutations predispose to an uncontrolled activation in the kidney and on blood cells and to secondary C3 deficiency. Plasma exchange has been used to correct for
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