跳转至内容
Merck
CN
所有图片(1)

文件

208880

Sigma-Aldrich

Anti-Calnexin, C-Terminal (575-593) Rabbit pAb

liquid, Calbiochem®

登录查看公司和协议定价

别名:
Anti-IP90, Anti-p88
UNSPSC代码:
12352203
NACRES:
NA.41

生物来源

rabbit

质量水平

抗体形式

purified antibody

抗体产品类型

primary antibodies

克隆

polyclonal

形式

liquid

包含

≤0.1% sodium azide as preservative

种属反应性

human, rabbit, mouse, rat, canine, guinea pig, bovine, hamster, monkey, sheep

制造商/商品名称

Calbiochem®

储存条件

OK to freeze
avoid repeated freeze/thaw cycles

同位素/亚型

IgG

运输

wet ice

储存温度

−20°C

靶向翻译后修饰

unmodified

一般描述

Anti-Calnexin, C-Terminal (575-593), rabbit polyclonal, recognizes the ~90 kDa calnexin protein. It is validated for Western blotting.
Protein A purified rabbit polyclonal antibody. Recognizes the ~90 kDa calnexin protein.
Recognizes the ~90 kDa calnexin protein.

免疫原

Canine
a synthetic peptide (AEEDEILNRSPRNRKPRRE) corresponding to amino acids 575-593 of canine calnexin, conjugated to KLH

应用

Immunoblotting (1 µg/ml)

警告

Toxicity: Standard Handling (A)

外形

In PBS, 50% glycerol, pH 7.2.

重悬

Following initial thaw, aliquot and freeze (-20°C).

分析说明

Positive Control
Heat-shocked HeLa cells

其他说明

Hammond, C., et al. 1994. J. Cell Biol. 126, 41.
Ou, W.-J., et al. 1993. Nature 364, 771.
David, V., et al. 1993. J. Biol. Chem.268, 9585.
Ahluwalia, N., et al. 1992. J. Biol. Chem.267, 10914.
Galvin, K., et al. 1992. Proc. Natl. Acad. Sci. USA89, 8452.
Hochstenbach, F., et al. 1992. Proc. Natl. Acad. Sci. USA89, 4734.
Wada, I., et al. 1991. J. Biol. Chem. 266, 19599.
Variables associated with assay conditions will dictate the optimal working dilution.

法律信息

CALBIOCHEM is a registered trademark of Merck KGaA, Darmstadt, Germany

WGK

WGK 1


分析证书(COA)

输入产品批号来搜索 分析证书(COA) 。批号可以在产品标签上"批“ (Lot或Batch)字后找到。

已有该产品?

在文件库中查找您最近购买产品的文档。

访问文档库

Martin Whitham et al.
The Journal of biological chemistry, 287(14), 10771-10779 (2012-02-22)
Exercise increases the expression of the prototypical myokine IL-6, but the precise mechanism by which this occurs has yet to be identified. To mimic exercise conditions, C2C12 myotubes were mechanically stimulated via electrical pulse stimulation (EPS). We compared the responses
Jin Zhang et al.
Virology journal, 9, 303-303 (2012-12-12)
Although the aggregation of PrPSc is thought to be crucial for the neuropathology of prion diseases, there is evidence in cultured cells and transgenic mice that neuronal death can be triggered by the accumulation of cytosolic PrPs, leading to the
Claudia M Wunderlich et al.
Journal of immunology (Baltimore, Md. : 1950), 188(9), 4141-4144 (2012-04-03)
Circulating IL-6 levels correlate with the severity of blood-stage malaria in humans and mouse models, but the impact of IL-6 classic signaling through membrane IL-6Rα, as well as IL-6 trans-signaling through soluble IL-6Rα, on the outcome of malaria has remained
Shami Kanekar et al.
Journal of neuroscience research, 87(6), 1424-1434 (2008-12-31)
One premise regarding the mechanism of injury-evoked neuroregeneration is that injured cells induce the release of neurotrophic factors to trigger neurogenesis. Extracellular purine nucleotides exert multiple neurotrophic actions in the central nervous system mediated via activation of purinergic receptors. However
Anita L Pinner et al.
Schizophrenia research, 177(1-3), 78-87 (2016-02-16)
Recent reports suggest abnormalities of neurotransmitter receptor trafficking, targeting, dendritic localization, recycling, and degradation in the brain in schizophrenia. We hypothesized that a potential explanation for these findings may be abnormal posttranslational modifications that influence intracellular targeting and trafficking of

我们的科学家团队拥有各种研究领域经验,包括生命科学、材料科学、化学合成、色谱、分析及许多其他领域.

联系技术服务部门