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Merck
CN
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文件

07-614

Sigma-Aldrich

抗表面活性剂蛋白B抗体

serum, Upstate®

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UNSPSC代码:
12352203
eCl@ss:
32160702
NACRES:
NA.41

生物来源

rabbit

质量水平

100

抗体形式

serum

抗体产品类型

primary antibodies

克隆

polyclonal

种属反应性

mouse, human

制造商/商品名称

Upstate®

技术

immunohistochemistry: suitable
western blot: suitable

同位素/亚型

IgG

UniProt登记号

P07988

运输

dry ice

靶向翻译后修饰

unmodified

基因信息

mouse ... Sftpd(20390)

特异性

表面活性蛋白B

免疫原

纯化的人表面活性蛋白B

应用

抗表面活性蛋白B抗体是用于IH & WB的抗表面活性蛋白B的抗体。
研究子类别
离子 & 运输渠道
研究类别
代谢

质量

通过免疫印迹对人细支气管灌洗液进行常规评估。

目标描述

8kDa

外形

兔抗血清，含有0.05%叠氮化钠，然后添加甘油至30%
血清

储存及稳定性

在-20°C下可保存2年

法律信息

UPSTATE is a registered trademark of Merck KGaA, Darmstadt, Germany

免责声明

除非我们的产品目录或产品附带的其他公司文档另有说明，否则我们的产品仅供研究使用，不得用于任何其他目的，包括但不限于未经授权的商业用途、体外诊断用途、离体或体内治疗用途或任何类型的消费或应用于人类或动物。

WGK

WGK 1

闪点(°F)

Not applicable

闪点(°C)

Not applicable

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Ralph Epaud et al.
American journal of respiratory cell and molecular biology, 28(3), 373-378 (2003-02-21)
Transgenic mice, in which the level of surfactant protein (SP)-B mature peptide varied 5.6-fold between SP-B(+/-) and SP-B-overexpressing lines (SP-B+/+/+), were used to test the hypothesis that SP-B protects against endotoxin-induced lung inflammation. Intratracheal administration of endotoxin resulted in significantly
Yan Liu et al.
Molecular reproduction and development, 84(8), 668-674 (2017-05-18)
Respiratory distress is a major cause of mortality in cloned neonatal animals, but its pathogenesis remains poorly understood. Here, we used necropsy and histology procedures to evaluate the lungs of cloned neonatal bovines dying of respiratory distress, finding incomplete lung
Lili Zhang et al.
Neoplasia (New York, N.Y.), 20(2), 207-217 (2018-01-15)
Risk stratification using molecular features could potentially help distinguish indolent from aggressive prostate cancer (PCa). Mutations in isocitrate dehydrogenase (IDH) acquire an abnormal enzymatic activity, resulting in the production of 2-hydroxyglutarate and alterations in cellular metabolism, histone modification, and DNA
L M Nogee et al.
American journal of respiratory and critical care medicine, 161(3 Pt 1), 973-981 (2000-03-11)
Inability to produce surfactant protein B (SP-B) causes fatal neonatal respiratory disease. A frame-shift mutation (121ins2) is the predominant but not exclusive cause of disease. To determine the range of mechanisms responsible for SP-B deficiency, both alleles from 32 affected
Surfactant chemical composition and biophysical activity in acute respiratory distress syndrome.
Gregory, T J, et al.
The Journal of Clinical Investigation, 88, 1976-1981 (1991)
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