推荐产品
生物来源
rabbit
质量水平
抗体形式
serum
抗体产品类型
primary antibodies
克隆
polyclonal
种属反应性
rat
制造商/商品名称
Upstate®
技术
western blot: suitable
同位素/亚型
IgG
NCBI登记号
UniProt登记号
运输
dry ice
靶向翻译后修饰
unmodified
基因信息
human ... AKAP1(8165)
特异性
AKAP 150
免疫原
一种对应于大鼠AKAP 150的428-449位氨基酸的肽(C-TTVGQAEEATVGQAEEATVGQA)
应用
抗AKAP 150抗体是可用于WB针对AKAP 150的抗体。
研究子类别
激酶&磷酸酶
RNA结合蛋白(RBP)
激酶&磷酸酶
RNA结合蛋白(RBP)
研究类别
信号传导
信号传导
质量
已通过免疫印迹对大鼠脑细胞溶质制剂进行常规评估
目标描述
150kDa
外形
0.1M Tris-甘氨酸(pH 7.4),0.15M NaCl,0.05%叠氮化钠,然后添加甘油至30%
抗血清
储存及稳定性
在-20°C下可保存2年
法律信息
UPSTATE is a registered trademark of Merck KGaA, Darmstadt, Germany
免责声明
除非我们的产品目录或产品附带的其他公司文档另有说明,否则我们的产品仅供研究使用,不得用于任何其他目的,包括但不限于未经授权的商业用途、体外诊断用途、离体或体内治疗用途或任何类型的消费或应用于人类或动物。
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储存分类代码
10 - Combustible liquids
WGK
WGK 1
AKAP signaling in reinstated cocaine seeking revealed by iTRAQ proteomic analysis.
The Journal of Neuroscience null
Science signaling, 10(463) (2017-01-26)
Hypercontractility of arterial myocytes and enhanced vascular tone during diabetes are, in part, attributed to the effects of increased glucose (hyperglycemia) on L-type CaV1.2 channels. In murine arterial myocytes, kinase-dependent mechanisms mediate the increase in CaV1.2 activity in response to
Addiction biology, 21(6), 1097-1112 (2016-10-26)
Alcohol use disorders (AUDs) are a major public health issue and produce enormous societal and economic burdens. Current Food and Drug Administration (FDA)-approved pharmacotherapies for treating AUDs suffer from deleterious side effects and are only effective in a subset of
Nature methods, 18(6), 669-677 (2021-06-02)
Single-molecule localization microscopy (SMLM) relies on the blinking behavior of a fluorophore, which is the stochastic switching between fluorescent and dark states. Blinking creates multiple localizations belonging to the same fluorophore, confounding quantitative analyses and interpretations. Here we present a
Cell communication and signaling : CCS, 22(1), 371-371 (2024-07-24)
Protein kinase A (PKA) enhances neurotransmission at the neuromuscular junction (NMJ), which is retrogradely regulated by nerve-induced muscle contraction to promote Acetylcholine (ACh) release through the phosphorylation of molecules involved in synaptic vesicle exocytosis (SNAP-25 and Synapsin-1). However, the molecular
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