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Merck
CN
所有图片(1)

主要文件

05-337

Sigma-Aldrich

Anti-MARCKS Antibody, clone 2F12

ascites fluid, clone 2F12, Upstate®

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About This Item

UNSPSC代码:
12352203
eCl@ss:
32160702
NACRES:
NA.41

生物来源

mouse

质量水平

抗体形式

ascites fluid

抗体产品类型

primary antibodies

克隆

2F12, monoclonal

种属反应性

bovine, human

制造商/商品名称

Upstate®

技术

immunoprecipitation (IP): suitable
western blot: suitable

同位素/亚型

IgG1

NCBI登记号

UniProt登记号

运输

wet ice

靶向翻译后修饰

unmodified

基因信息

human ... MARCKS(4082)

特异性

MARCKS

免疫原

Purified recombinant human MARCKS; Epitope mapped to the carboxyl-terminal.

应用

Anti-MARCKS Antibody, clone 2F12 detects level of MARCKS & has been published & validated for use in IP & WB.
Research Category
Signaling
Research Sub Category
Cytoskeletal Signaling

质量

routinely evaluated by immunoblot on bovine brain extract

目标描述

none given

外形

Ascites
mouse ascites containing 0.05% sodium azide

储存及稳定性

6 months at -20°C

法律信息

UPSTATE is a registered trademark of Merck KGaA, Darmstadt, Germany

免责声明

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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储存分类代码

12 - Non Combustible Liquids

WGK

nwg

闪点(°F)

Not applicable

闪点(°C)

Not applicable


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20-Hydroxyeicosatetraenoic acid-induced vasoconstriction and inhibition of potassium current in cerebral vascular smooth muscle is dependent on activation of protein kinase C
Lange, A., et al
The Journal of Biological Chemistry, 272, 27345-27352 (1997)
Shijing Fang et al.
American journal of physiology. Lung cellular and molecular physiology, 304(8), L511-L518 (2013-02-05)
Myristoylated alanine-rich C kinase substrate (MARCKS) protein has been recognized as a key regulatory molecule controlling mucin secretion by airway epithelial cells in vitro and in vivo. We recently showed that two intracellular chaperones, heat shock protein 70 (HSP70) and
Jin-Ah Park et al.
The American journal of pathology, 171(6), 1822-1830 (2007-12-07)
Mucin hypersecretion is a major pathological feature of many respiratory diseases, yet cellular mechanisms regulating secretion of mucin have not been fully elucidated. Previously, we reported that mucin hypersecretion induced by human neutrophil elastase involves activation of protein kinase C

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