检测方案
>99% (TLC)
形式
powder
包装
pkg of 2 × 100 mg (850458P-200mg)
pkg of 1 × 25 mg (850458P-25mg)
制造商/商品名称
Avanti Research™ - A Croda Brand 850458P
脂质类型
cardiolipins
phospholipids
运输
dry ice
储存温度
−20°C
SMILES字符串
[O-]P(OCC[N+](C)(C)C)(OC[C@]([H])(OC(CCCCCCC/C=C\C/C=C\CCCCC)=O)COC(CCCCCCCCCCCCCCC)=O)=O
一般描述
16:0-18:2 PC或1-棕榈酰基-2-亚油酰基-sn-甘油-3-磷酸胆碱是一种甘油磷脂，在甘油主链的sn-1和sn-2位置分别被棕榈酸和亚麻酸取代。
应用
16:0-18:2 PC或1-棕榈酰基-2-亚油酰基-sn-甘油-3-磷酸胆碱
- ：已用于脂质体的标准无细胞翻译
- 已用于提取血清脂质和过氧化物酶体增殖物激活受体-δ（PPAR-δ）测定
- 可用于研究磷脂酰胆碱（PCs）对受损间皮的润滑作用
包装
5 mL透明玻璃密封安瓿（850458P-200mg）
5 mL透明玻璃密封安瓿（850458P-25mg）
法律信息
Avanti Research is a trademark of Avanti Polar Lipids, LLC
储存分类代码
11 - Combustible Solids
Francesca Bodega et al.
Respiratory physiology & neurobiology, 203, 116-120 (2014-08-17)
Effect of time and phosphatidylcholines (PCs) on lubrication of damaged mesothelium has been investigated. Marked increase in coefficient of kinetic friction (μ) of pleural specimens after mesothelial blotting and rewetting decreased by 23.4±3.5%, 41.8±3.8%, and 40.5±2.7% after 30min, 1h, and
Giulia Coliva et al.
Molecules (Basel, Switzerland), 25(8) (2020-04-25)
Free radical driven lipid peroxidation is a chain reaction which can lead to oxidative degradation of biological membranes. Propagation vs. termination rates of peroxidation in biological membranes are determined by a variety of factors including fatty acyl chain composition, presence
Lindsay Kosack et al.
Scientific reports, 7(1), 11289-11289 (2017-09-14)
Lipid metabolism is increasingly being appreciated to affect immunoregulation, inflammation and pathology. In this study we found that mice infected with lymphocytic choriomeningitis virus (LCMV) exhibit global perturbations of circulating serum lipids. Mice lacking the lipid-sensing surface receptor triggering receptor
Eikan Mishima et al.
Journal of the American Society of Nephrology : JASN, 31(2), 280-296 (2019-11-27)
Ferroptosis, nonapoptotic cell death mediated by free radical reactions and driven by the oxidative degradation of lipids, is a therapeutic target because of its role in organ damage, including AKI. Ferroptosis-causing radicals that are targeted by ferroptosis suppressors have not
John K Eaton et al.
Nature chemical biology, 16(5), 497-506 (2020-04-02)
We recently described glutathione peroxidase 4 (GPX4) as a promising target for killing therapy-resistant cancer cells via ferroptosis. The onset of therapy resistance by multiple types of treatment results in a stable cell state marked by high levels of polyunsaturated
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