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经验公式(希尔记法):
C8H5F3O2S
化学文摘社编号:
分子量:
222.18
UNSPSC Code:
12352100
NACRES:
NA.22
PubChem Substance ID:
EC Number:
206-316-7
Beilstein/REAXYS Number:
168645
MDL number:
Assay:
99%
InChI key
TXBBUSUXYMIVOS-UHFFFAOYSA-N
InChI
1S/C8H5F3O2S/c9-8(10,11)7(13)4-5(12)6-2-1-3-14-6/h1-3H,4H2
SMILES string
FC(F)(F)C(=O)CC(=O)c1cccs1
assay
99%
bp
96-98 °C/8 mmHg (lit.)
Quality Level
mp
40-44 °C (lit.)
storage temp.
2-8°C
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Application
用于锕系元素和镧系元素的测定。
存储类别
11 - Combustible Solids
wgk
WGK 3
flash_point_f
233.6 °F - closed cup
flash_point_c
112 °C - closed cup
ppe
dust mask type N95 (US), Eyeshields, Gloves
Jeffery, G.H. et al.
Vogel's Textbook of Quantitative Chemical Analysis, 170-170 (1989)
Vladimir A Kokorekin et al.
Molecules (Basel, Switzerland), 25(18) (2020-09-17)
In this article, we demonstrate how an original effective "metal-free" and "chromatography-free" route for the synthesis of 3-thiocyanatopyrazolo[1,5-a]pyrimidines has been developed. It is based on electrooxidative (anodic) C-H thiocyanation of 5-aminopyrazoles by thiocyanate ion leading to 4-thiocyanato-5-aminopyrazoles (stage 1, yields
Pin-Zhu Qin et al.
Talanta, 80(2), 991-995 (2009-10-20)
A two-probe tandem DNA hybridization assay including capture DNA(1), probe DNA(2), and target DNA(3) was prepared. The long-lived luminescent europium complex doped nanoparticles (NPs) were used as the biomarker. The complex included in the particle was Eu(TTA)(3)(5-NH(2)-phen)-IgG (ETN-IgG), the europium
Hae-Ok Byun et al.
Journal of cellular biochemistry, 104(5), 1747-1759 (2008-04-09)
Mitochondrial complex II defect has recently been implicated in cellular senescence and in the ageing process of which a critical phenotype is retardation and arrest of cellular growth. However, the underlying mechanisms of how complex II defect affects cellular growth
Stefan Dröse et al.
Molecular pharmacology, 79(5), 814-822 (2011-02-01)
In this study, we have analyzed the effect of different cardioprotective complex II inhibitors on the mitochondrial production of reactive oxygen species (ROS) because ROS seem to be essential for signaling during preconditioning to prevent ischemia/reperfusion injury. Despite different binding
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