推荐产品
方案
99.5%
表单
wire
自燃温度
1400 °F
制造商/商品名称
Goodfellow 819-232-38
电阻率
2.6548 μΩ-cm
长度 × 直径
25 m × 0.25 mm
沸点
2460 °C (lit.)
mp
660.37 °C (lit.)
密度
2.7 g/mL at 25 °C (lit.)
SMILES字符串
[Al]
InChI
1S/Al
InChI key
XAGFODPZIPBFFR-UHFFFAOYSA-N
一般描述
For updated SDS information please visit www.goodfellow.com.
法律信息
Product of Goodfellow
储存分类代码
13 - Non Combustible Solids
WGK
WGK 3
闪点(°F)
Not applicable
闪点(°C)
Not applicable
法规信息
新产品
Stephen C Bondy
Toxicology, 315, 1-7 (2013-11-06)
Aluminum is one of the most common metal elements in the earth's crust. It is not an essential element for life and has commonly been thought of as a rather inert and insoluble mineral. Therefore, it has often been regarded
C A Shaw et al.
Immunologic research, 56(2-3), 304-316 (2013-04-24)
We have examined the neurotoxicity of aluminum in humans and animals under various conditions, following different routes of administration, and provide an overview of the various associated disease states. The literature demonstrates clearly negative impacts of aluminum on the nervous
M A Santos et al.
Current medicinal chemistry, 19(17), 2773-2793 (2012-03-30)
This review is focused on recent developments on hydroxypyri(mi)dines, as aluminum and actinide chelating agents to combat the toxicity due to accumulations of these metal ions in human body resulting from excessive metal exposure. After a brief update revision of
Maire E Percy et al.
Journal of inorganic biochemistry, 105(11), 1505-1512 (2011-11-22)
In 1991, treatment with low dose intramuscular desferrioxamine (DFO), a trivalent chelator that can remove excessive iron and/or aluminum from the body, was reported to slow the progression of Alzheimer's disease (AD) by a factor of two. Twenty years later
J R Walton
Journal of Alzheimer's disease : JAD, 35(1), 7-43 (2013-02-06)
The neuroanatomic specificity with which Alzheimer's disease (AD) progresses could provide clues to AD etiopathology. Magnetic resonance imaging studies of AD clinical progression have confirmed general conclusions from earlier studies of AD neuropathological progression wherein neurofibrillary tangle pathology was observed
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