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Merck
CN

GF69970427

foil, 300x300mm, thickness 0.25mm, hard, 99.5%

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About This Item

线性分子式:
Fe
CAS号:
分子量:
55.85
MDL编号:
UNSPSC代码:
12141721
PubChem化学物质编号:
NACRES:
NA.23

检测方案

99.5%

形式

foil

制造商/商品名称

Goodfellow 699-704-27

电阻率

9.71 μΩ-cm

尺寸 × 厚度

300x300 mm × 0.25 mm

bp

2750 °C (lit.)

mp

1535 °C (lit.)

密度

7.86 g/mL at 25 °C (lit.)

SMILES字符串

[Fe]

InChI

1S/Fe

InChI key

XEEYBQQBJWHFJM-UHFFFAOYSA-N

一般描述

For updated SDS information please visit www.goodfellow.com.

法律信息

Product of Goodfellow

法规信息

新产品

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Arnold L Demain et al.
Applied microbiology and biotechnology, 73(1), 55-59 (2006-04-20)
When tetanus toxin is made by fermentation with Clostridium tetani, the traditional source of iron is an insoluble preparation called reduced iron powder. This material removes oxygen from the system by forming FeO(2) (rust). When inoculated in a newly developed
Jun-Won Jang et al.
Water science and technology : a journal of the International Association on Water Pollution Research, 59(12), 2503-2507 (2009-06-23)
Zero valent iron has been successfully used for the degradation of a wide range of contaminants. However, this reaction of using ZVI particle produces a large quantity of iron sludge. To solve the problem, we report the synthesis of self-organized
50 years ago in the Journal of Pediatrics: Iron metabolism in premature infants: II. Prevention of iron deficiency.
Pamela J Kling
The Journal of pediatrics, 164(4), 768-768 (2014-03-22)
Laura M van Staalduinen et al.
Proceedings of the National Academy of Sciences of the United States of America, 111(14), 5171-5176 (2014-04-08)
The enzymes PhnY and PhnZ comprise an oxidative catabolic pathway that enables marine bacteria to use 2-aminoethylphosphonic acid as a source of inorganic phosphate. PhnZ is notable for catalyzing the oxidative cleavage of a carbon-phosphorus bond using Fe(II) and dioxygen
Thomas A Russo et al.
Infection and immunity, 82(6), 2356-2367 (2014-03-26)
Hypervirulent (hypermucoviscous) Klebsiella pneumoniae (hvKP) strains are an emerging variant of "classical" K. pneumoniae (cKP) that cause organ and life-threatening infection in healthy individuals. An understanding of hvKP-specific virulence mechanisms that enabled evolution from cKP is limited. Observations by our

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