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Merck
CN

GF66605856

foil, 100x100mm, thickness 0.125mm, as rolled, 99.99+%

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线性分子式:
Fe
化学文摘社编号:
分子量:
55.85
NACRES:
NA.23
PubChem Substance ID:
UNSPSC Code:
12141721
MDL number:
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产品名称

铁, foil, 100x100mm, thickness 0.125mm, as rolled, 99.99+%

InChI

1S/Fe

SMILES string

[Fe]

InChI key

XEEYBQQBJWHFJM-UHFFFAOYSA-N

assay

99.99%

form

foil

manufacturer/tradename

Goodfellow 666-058-56

resistivity

9.71 μΩ-cm

size × thickness

100x100 mm × 0.125 mm

bp

2750 °C (lit.)

mp

1535 °C (lit.)

density

7.86 g/mL at 25 °C (lit.)

General description

For updated SDS information please visit www.goodfellow.com.

Legal Information

Product of Goodfellow

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Qiu et al.
Physical review letters, 85(7), 1492-1495 (2000-09-06)
FeF (2) films are grown by the reaction of XeF (2) and SeF (6) with iron foil. The growth initially follows the Mott-Cabrera parabolic rate law, indicating that the process is diffusion limited. At a certain film thickness, however, the
Arnold L Demain et al.
Applied microbiology and biotechnology, 73(1), 55-59 (2006-04-20)
When tetanus toxin is made by fermentation with Clostridium tetani, the traditional source of iron is an insoluble preparation called reduced iron powder. This material removes oxygen from the system by forming FeO(2) (rust). When inoculated in a newly developed
Thomas A Russo et al.
Infection and immunity, 82(6), 2356-2367 (2014-03-26)
Hypervirulent (hypermucoviscous) Klebsiella pneumoniae (hvKP) strains are an emerging variant of "classical" K. pneumoniae (cKP) that cause organ and life-threatening infection in healthy individuals. An understanding of hvKP-specific virulence mechanisms that enabled evolution from cKP is limited. Observations by our
Hypoferremia of infection: a double-edged sword?
Kristen L Lokken et al.
Nature medicine, 20(4), 335-337 (2014-04-09)
Eunna Choi et al.
Infection and immunity, 82(6), 2203-2210 (2014-03-20)
In order to survive inside macrophages, Salmonella produces a series of proteins encoded by genes within Salmonella pathogenicity island 2 (SPI-2). In the present study, we report that Fur, a central regulator of iron utilization, negatively controls the expression of

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