推荐产品
方案
99.999%
表单
rod
制造商/商品名称
Goodfellow 274-298-91
电阻率
1.673 μΩ-cm, 20°C
长度 × 直径
100 mm × 6.35 mm
沸点
2567 °C (lit.)
mp
1083.4 °C (lit.)
密度
8.94 g/mL at 25 °C (lit.)
SMILES字符串
[Cu]
InChI
1S/Cu
InChI key
RYGMFSIKBFXOCR-UHFFFAOYSA-N
一般描述
For updated SDS information please visit www.goodfellow.com.
法律信息
Product of Goodfellow
储存分类代码
13 - Non Combustible Solids
WGK
WGK 2
闪点(°F)
Not applicable
闪点(°C)
Not applicable
法规信息
新产品
Donita C Brady et al.
Nature, 509(7501), 492-496 (2014-04-11)
The BRAF kinase is mutated, typically Val 600→Glu (V600E), to induce an active oncogenic state in a large fraction of melanomas, thyroid cancers, hairy cell leukaemias and, to a smaller extent, a wide spectrum of other cancers. BRAF(V600E) phosphorylates and
Hiroki Serizawa et al.
Organic letters, 16(13), 3456-3459 (2014-06-14)
The direct synthesis of pentafluoroethyl copper (CuC2F5) from a cuprate reagent and ethyl pentafluoropropionate as one of the most economical and useful pentafluoroethyl sources was accomplished. The advantages of this method are; all the reagents employed are low-cost and operationally
Gwilherm Evano et al.
Natural product reports, 30(12), 1467-1489 (2013-10-25)
Copper-catalyzed Ullmann-Goldberg-type cross-coupling reactions have undergone nothing short of a renaissance over the last decade and an impressive number of procedures are now available for the formation of C-N, C-O and C-S bonds with remarkable efficiencies and surgical precision. These
Nathalie Adam et al.
Environmental pollution (Barking, Essex : 1987), 194, 130-137 (2014-08-12)
In this study the uptake of ZnO and CuO nanoparticles by Daphnia magna was tested. Daphnids were exposed during 48 h to acute concentrations of the nanoparticles and corresponding metal salts. The Daphnia zinc and copper concentration was measured and the
Lelita T Braiterman et al.
Proceedings of the National Academy of Sciences of the United States of America, 111(14), E1364-E1373 (2014-04-08)
Wilson disease (WD) is a monogenic autosomal-recessive disorder of copper accumulation that leads to liver failure and/or neurological deficits. WD is caused by mutations in ATP7B, a transporter that loads Cu(I) onto newly synthesized cupro-enzymes in the trans-Golgi network (TGN)
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