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Merck
CN

A68203

6-氨基烟酰胺

99%

别名:

6AN, 6-氨基-3-吡啶甲酰胺

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关于此项目

经验公式(希尔记法):
C6H7N3O
化学文摘社编号:
分子量:
137.14
UNSPSC Code:
12352100
NACRES:
NA.22
PubChem Substance ID:
EC Number:
206-349-7
Beilstein/REAXYS Number:
116042
MDL number:
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产品名称

6-氨基烟酰胺, 99%

InChI key

ZLWYEPMDOUQDBW-UHFFFAOYSA-N

InChI

1S/C6H7N3O/c7-5-2-1-4(3-9-5)6(8)10/h1-3H,(H2,7,9)(H2,8,10)

SMILES string

NC(=O)c1ccc(N)nc1

assay

99%

form

powder

mp

245-248 °C (lit.)

Quality Level

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Application

6-氨基烟酰胺可作为以下应用的反应物:
  • 合成6-取代的咪唑并[1,2-a吡啶,此产物有可能作为化学治疗药物。
  • 脱水 N-单苄基化过程。

General description

6-氨基烟酰胺是氨基吡啶,为特异性戊糖抑制剂,抑制 NADP产生。

pictograms

Health hazard

signalword

Danger

hcodes

Hazard Classifications

Repr. 1B

存储类别

6.1C - Combustible acute toxic Cat.3 / toxic compounds or compounds which causing chronic effects

wgk

WGK 3

ppe

Eyeshields, Gloves, type P2 (EN 143) respirator cartridges


历史批次信息供参考:

分析证书(COA)

Lot/Batch Number

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A borrowing hydrogen methodology: palladium-catalyzed dehydrative N-benzylation of 2-aminopyridines in water.
Hikawa H, et al.
Green Chemistry, 20(13), 3044-3049 (2018)
L Poulain et al.
Leukemia, 31(11), 2326-2335 (2017-03-11)
Alterations in metabolic activities are cancer hallmarks that offer a wide range of new therapeutic opportunities. Here we decipher the interplay between mTORC1 activity and glucose metabolism in acute myeloid leukemia (AML). We show that mTORC1 signaling that is constantly
6-Substituted imidazo [1, 2-a] pyridines: Synthesis and biological activity against colon cancer cell lines HT-29 and Caco-2.
Dahan-Farkas N, et al.
European Journal of Medicinal Chemistry, 46(9), 4573-4583 (2011)
Electrocatalytic synthesis of 6-aminonicotinic acid at silver cathodes under mild conditions.
Gennaro A, et al.
Electrochemical Communications, 6(7), 627-631 (2004)
Jorge Domínguez-Andrés et al.
PLoS pathogens, 13(9), e1006632-e1006632 (2017-09-19)
Monocytes are innate immune cells that play a pivotal role in antifungal immunity, but little is known regarding the cellular metabolic events that regulate their function during infection. Using complementary transcriptomic and immunological studies in human primary monocytes, we show

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