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About This Item
线性分子式:
NH2CH2CH2OSO3H
CAS号:
分子量:
141.15
Beilstein:
1704079
EC 号:
MDL编号:
UNSPSC代码:
12352100
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方案
97%
mp
277 °C (dec.) (lit.)
SMILES字符串
NCCOS(O)(=O)=O
InChI
1S/C2H7NO4S/c3-1-2-7-8(4,5)6/h1-3H2,(H,4,5,6)
InChI key
WSYUEVRAMDSJKL-UHFFFAOYSA-N
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警示用语：
Warning
危险分类
Acute Tox. 4 Oral - Eye Irrit. 2 - Skin Irrit. 2 - STOT SE 3
储存分类代码
13 - Non Combustible Solids
WGK
WGK 3
闪点(°F)
Not applicable
闪点(°C)
Not applicable
个人防护装备
dust mask type N95 (US), Eyeshields, Gloves
法规信息
新产品
S Lindgren et al.
British journal of pharmacology, 91(3), 617-625 (1987-07-01)
Slice preparations of rat cuneate nucleus were used for studies on the gamma-aminobutyric acid GABAA-receptor complex following chronic and acute pretreatment with GABA-alpha-ketoglutarate aminotransferase (GABA-T) inhibitors. The whole brain GABA concentration was significantly increased 2.9 fold and 2.6 fold following
R A John et al.
Biochemical pharmacology, 36(9), 1467-1473 (1987-05-01)
Two "suicide" inhibitors of GABA-aminotransferase which are known to raise the concentration of GABA in vivo and to have anti-convulsant properties, have been compared for the extent to which they produce micro-vacuoles in the brains of rats. The compounds gamma-vinyl-GABA
A Fernández-Guasti et al.
Pharmacology, biochemistry, and behavior, 24(4), 1065-1070 (1986-04-01)
Drugs affecting the GABAergic transmission were injected into the medial preoptic anterior hypothalamic area (MPOA) and the masculine sexual behavior analyzed. Antagonizing GABAergic neurotransmission by (+) bicuculline methiodide (30 ng/cannula), or 3-mercaptopropionic acid (10 or 20 micrograms/cannula) resulted in a
S Nagaki et al.
Life sciences, 46(22), 1587-1595 (1990-01-01)
Immunoreactive somatostatin (IR-SRIF) and gamma-aminobutyric acid (GABA) contents in the rat brain were investigated to study chronic effects of the treatment with anticonvulsants, carbamazepine (CBZ), valproic acid (VPA) and phenytoin (PHT). Decreased IR-SRIF levels were found in several brain regions
H Hodges et al.
Physiology & behavior, 41(3), 257-264 (1987-01-01)
Studies have shown that benzodiazepines (BZs) both disrupt discrimination and increase resistance to punishment. Using a delayed response task, we provide evidence that effects of BZs on discrimination cannot be fully explained by deficits in either short or long term
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