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经验公式(希尔记法):
C7H7NO
化学文摘社编号:
分子量:
121.14
UNSPSC Code:
12352100
NACRES:
NA.22
PubChem Substance ID:
EC Number:
206-496-7
Beilstein/REAXYS Number:
107751
MDL number:
Assay:
98%
Form:
liquid
InChI
1S/C7H7NO/c1-6(9)7-3-2-4-8-5-7/h2-5H,1H3
InChI key
WEGYGNROSJDEIW-UHFFFAOYSA-N
SMILES string
CC(=O)c1cccnc1
assay
98%
form
liquid
Quality Level
bp
220 °C (lit.)
mp
11-13 °C (lit.)
density
1.102 g/mL at 25 °C (lit.)
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signalword
Danger
hcodes
Hazard Classifications
Acute Tox. 3 Oral - Skin Irrit. 2
存储类别
6.1C - Combustible acute toxic Cat.3 / toxic compounds or compounds which causing chronic effects
wgk
WGK 1
flash_point_f
219.2 °F - closed cup
flash_point_c
104 °C - closed cup
ppe
Eyeshields, Faceshields, Gloves, type ABEK (EN14387) respirator filter
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Neurodegenerative lesions induce sprouting of new collaterals from surviving axons, but the extent to which this form of axonal remodelling alters brain functional structure remains unclear. To understand how collateral sprouting proceeds in the adult brain, we imaged post-lesion sprouting
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The essential coenzyme nicotinamide adenine dinucleotide (NAD+) plays important roles in metabolic reactions and cell regulation in all organisms. Bacteria, fungi, plants, and animals use different pathways to synthesize NAD+. Our molecular and genetic data demonstrate that in the unicellular
Iran Goudarzi et al.
European journal of pharmacology, 642(1-3), 56-65 (2010-07-20)
Electrophysiological dysfunction of Purkinje cells causes cerebellar ataxia. Recent studies indicated that 4-aminopyridine (4-AP) can prevent the attacks in patients with episodic ataxia type 2. However, the cellular mechanism(s) by which 4-AP might be beneficial for the improvement of motor
Mahyar Janahmadi et al.
Neurotoxicology, 30(3), 393-402 (2009-05-16)
Riluzole has been shown to possess neuroprotective effects in a variety of neurological and animal model of diseases, including motor diseases. However, the mechanism(s) by which riluzole preserves the intrinsic electrophysiological characteristics of neuronal membrane has not been fully delineated.
T M Kuchmerovskaia et al.
Voprosy meditsinskoi khimii, 48(3), 264-270 (2002-09-24)
The effect of administration of 3-acetylpyridine (antivitamin B3), on rat brain serotoninergic and NAD receptor systems was investigated. The injection of 3-acetylpyridine to rats caused a decrease of NAD and serotonin content in the brain and changes in [U-14C]NAD binding
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