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Merck
CN
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安全信息

690228

Sigma-Aldrich

N,N-双[3-(甲氨基)丙基]甲胺

98%

别名:

N-甲基-3,3′-双(甲氨基)二丙胺, N-甲基-N,N-双[3-(甲氨基)丙基]胺, 甲基双(3-甲基氨丙基)胺

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About This Item

线性分子式:
CH3N(CH2CH2CH2NHCH3)2
CAS号:
123-70-6
分子量:
173.30
Beilstein:
6711228
EC 号:
204-645-0
MDL编号:
MFCD00014857
UNSPSC代码:
12352100
PubChem化学物质编号:
329761321
NACRES:
NA.22

方案

≥97.5% (GC)
97.5-102.5% (with HClO4, T)
98%

表单

liquid

杂质

≤1% water

SMILES字符串

CNCCCN(C)CCCNC

InChI

1S/C9H23N3/c1-10-6-4-8-12(3)9-5-7-11-2/h10-11H,4-9H2,1-3H3

InChI key

PQYGBJHVVYREGU-UHFFFAOYSA-N

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象形图

Corrosion
GHS05

警示用语：

Danger

危险声明

H314

危险分类

Skin Corr. 1B

储存分类代码

8A - Combustible corrosive hazardous materials

WGK

WGK 3

闪点(°F)

Not applicable

闪点(°C)

Not applicable

个人防护装备

Faceshields, Gloves, Goggles, type ABEK (EN14387) respirator filter

法规信息

新产品

历史批次信息供参考:

分析证书(COA)

Lot/Batch Number
1414612V
1414612

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S Levine et al.
Journal of immunological methods, 54(3), 355-360 (1982-11-12)
In the past, the lesions of experimental allergic encephalomyelitis (EAE) have been induced to localize around brain tissue damaged by anoxia or direct physical or chemical attack. The procedure for producing the requisite antecedent brain injury has been simplified by
D Nochlin et al.
Journal of neuropathology and experimental neurology, 41(2), 233-240 (1982-03-01)
The administration of an aliphatic triamine, 3,3'-methylimino-bis-(N-methylpropylamine), was used to produce non-necrotizing lesions in the hypothalamus and medulla oblongata of rats. The distribution of the lesions in the hypothalamus was similar to that produced by goldthioglucose, and consisted ultrastructurally of
Gold thioglucose, mimpa and lesions of the ventromedial hypothalamus.
D F Brown
Journal of neuropathology and experimental neurology, 43(4), 455-457 (1984-07-01)
S Levine et al.
Brain research, 242(2), 219-225 (1982-06-24)
An aliphatic triamine has been reported to cause lesions in rats in the vicinity of the area postrema and the median eminence of the hypothalamus, sites known to lack a blood-brain barrier. The present study revealed that some of the
J C Lyons et al.
Journal of neuropathology and experimental neurology, 41(1), 45-53 (1982-01-01)
The clinical and pathologic features of two brothers with the adult form of Gaucher's disease, both of whom developed cerebral glioblastoma multiforme, are presented. Neither patient had a long-standing neurologic disorder, and morphologic evidence of nervous system glucocerebroside storage was
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