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同位素纯度
95 atom % 11B
形式
solid
质量偏移
depleted
SMILES字符串
[11B]
InChI
1S/B/i1+0
InChI key
ZOXJGFHDIHLPTG-IGMARMGPSA-N
包装
This product may be available from bulk stock and can be packaged on demand. For information on pricing, availability and packaging, please contact Stable Isotopes Customer Service.
警示用语：
Warning
危险声明
危险分类
Acute Tox. 4 Oral - Aquatic Chronic 3
WGK
WGK 3
闪点(°F)
Not applicable
闪点(°C)
Not applicable
Chathura D Suraweera et al.
Cell death & disease, 9(10), 967-967 (2018-09-22)
Bcl-2 family proteins play a crucial role in regulating apoptosis, a process critical for development, eliminating damaged or infected cells, host-pathogen interactions and in disease. Dysregulation of Bcl-2 proteins elicits an expansive cell survival mechanism promoting cell migration, invasion and
John C Rose et al.
Nature methods, 14(9), 891-896 (2017-07-25)
We developed a chemically inducible Cas9 (ciCas9) and a droplet digital PCR assay for double-strand breaks (DSB-ddPCR) to investigate the kinetics of Cas9-mediated generation and repair of DSBs in cells. ciCas9 is a rapidly activated, single-component Cas9 variant engineered by
Michael Fricker et al.
Physiological reviews, 98(2), 813-880 (2018-03-01)
Neuronal cell death occurs extensively during development and pathology, where it is especially important because of the limited capacity of adult neurons to proliferate or be replaced. The concept of cell death used to be simple as there were just
S Matt Gilbert et al.
Scientific reports, 7(1), 15096-15096 (2017-11-10)
We demonstrate the fabrication of individual nanopores in hexagonal boron nitride (h-BN) with atomically precise control of the pore shape and size. Previous methods of pore production in other 2D materials typically create pores with irregular geometry and imprecise diameters.
Yulia Kushnareva et al.
PLoS biology, 10(9), e1001394-e1001394 (2012-10-11)
Bax/Bak-mediated mitochondrial outer membrane permeabilization (MOMP) is essential for "intrinsic" apoptotic cell death. Published studies used synthetic liposomes to reveal an intrinsic pore-forming activity of Bax, but it is unclear how other mitochondrial outer membrane (MOM) proteins might facilitate this
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