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Merck
CN

448257

Sigma-Aldrich

5-(甲硫基)-1H-四唑

97%

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经验公式(希尔记法):
C2H4N4S
CAS号:
分子量:
116.14
MDL编号:
UNSPSC代码:
12352100
PubChem化学物质编号:
NACRES:
NA.22

检测方案

97%

形式

solid

mp

149-152 °C (lit.)

SMILES字符串

CSc1nnn[nH]1

InChI

1S/C2H4N4S/c1-7-2-3-5-6-4-2/h1H3,(H,3,4,5,6)

InChI key

ZBXNFTFKKOSPLD-UHFFFAOYSA-N

象形图

Exclamation mark

警示用语:

Warning

危险声明

危险分类

Eye Irrit. 2 - Skin Irrit. 2 - STOT SE 3

靶器官

Respiratory system

WGK

WGK 3

闪点(°F)

Not applicable

闪点(°C)

Not applicable

个人防护装备

dust mask type N95 (US), Eyeshields, Gloves

法规信息

监管及禁止进口产品

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The Effectivity of 1H-Triazoles and-Tetrazoles as Activators in Acid-Catalyzed Phosphoramidite Alcoholysis.
Nurminen E, et al.
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International journal of biomaterials, 2009, 512417-512417 (2010-02-05)
A novel bioanalogue hydroxyapatite (HAp)/chitosan phosphate (CSP) nanocomposite has been synthesized by a solution-based chemical methodology with varying HAp contents from 10 to 60% (w/w). The interfacial bonding interaction between HAp and CSP has been investigated through Fourier transform infrared
Laura Tapella et al.
Pituitary, 20(3), 311-318 (2016-11-18)
Endocrine disruptors are known to modulate a variety of endocrine functions and increase the risk for neoplasia. Epidemiological data reported increased prevalence of pituitary tumors in high industrial areas while genotyping studies showed that mutations in the aryl hydrocarbon receptor
Jing Cheng et al.
Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology, 46(6), 2587-2600 (2018-05-15)
The present study aimed to detect the expression of miR-449a and investigate the effect of miR-449a on cell injury in cardiomyocytes subjected to hypoxia/ reoxygenation (H/R) and its underlying mechanisms. The expression of miR-449a was determined using reverse transcription-polymerase chain
B Guan et al.
Acta endocrinologica (Bucharest, Romania : 2005), 15(2), 158-164 (2019-09-12)
Endoplasmic reticulum stress (ERS) is suspected as an important factor in the initiation of insulin resistance. To explore the effects of exendin-4 (Ex-4) on the endoplasmic reticulum stress (ERS)-mediated insulin resistance in 3T3-L1 adipocytes. In our study, 3T3-L1 adipocytes were

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