选择尺寸
500 ML
CN¥434.58
1 L
CN¥700.66
6 X 500 ML
CN¥2,085.93
24 X 500 ML
CN¥7,822.27
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500 ML
CN¥434.58
1 L
CN¥700.66
6 X 500 ML
CN¥2,085.93
24 X 500 ML
CN¥7,822.27
About This Item
经验公式（希尔记法）:
Ni3S2
CAS号:
分子量:
240.21
EC 号:
MDL编号:
UNSPSC代码:
12161600
PubChem化学物质编号:
NACRES:
NA.23
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方案
99.7% trace metals basis
表单
powder
反应适用性
reagent type: catalyst
core: nickel
粒径
−200 mesh
密度
5.8 g/mL at 25 °C (lit.)
应用
battery manufacturing
SMILES字符串
[Ni]=S=[Ni]=S=[Ni]
InChI
1S/3Ni.2S
InChI key
YGHCWPXPAHSSNA-UHFFFAOYSA-N
1 of 4
此商品 | 116807 | 541281 | 644927 |
|---|---|---|---|
| reaction suitability reagent type: oxidant | reaction suitability - | reaction suitability - | reaction suitability - |
| Quality Level 200 | Quality Level 100 | Quality Level 100 | Quality Level - |
| mp 69-71 °C (lit.) | mp 35-38 °C (lit.) | mp 128-132 °C (lit.) | mp 236-240 °C (lit.) |
| storage temp. 2-8°C | storage temp. - | storage temp. - | storage temp. - |
| form solid | form solid | form - | form solid |
一般描述
Major phase is Ni3S2. Product may also include a small amount of sulfur-rich phase. Nickel Sulfide (Ni3S2) exists as a heazlewoodite mineral in nature. The structure of Ni3S2 is hombohedral. Raman spectral analysis of Ni3S2 has been reported.[1]
应用
Nickel sulfide was implanted in mice, to induce the growth of malignant fibrous histiocytomas.[2]
包装
Packaged in glass bottles
警示用语：
Danger
危险分类
Acute Tox. 3 Inhalation - Aquatic Acute 1 - Aquatic Chronic 1 - Carc. 1A Inhalation - Muta. 2 - Skin Sens. 1 - STOT RE 1 Inhalation
靶器官
Lungs
储存分类代码
6.1D - Non-combustible acute toxic Cat.3 / toxic hazardous materials or hazardous materials causing chronic effects
WGK
WGK 3
闪点(°F)
Not applicable
闪点(°C)
Not applicable
个人防护装备
Eyeshields, Faceshields, Gloves, type P3 (EN 143) respirator cartridges
Aaron Barchowsky et al.
The Journal of biological chemistry, 277(27), 24225-24231 (2002-04-30)
Inhalation of particulate nickel subsulfide (Ni3S2) causes chronic active inflammation and fibrosis of the lungs. However, the mechanisms for these effects are not well understood. Therefore, cell culture experiments with BEAS-2B human airway epithelial cells were conducted to test the
Joseph R Landolph et al.
Environmental health perspectives, 110 Suppl 5, 845-850 (2002-11-12)
In the past, exposure of workers to mixtures of soluble and insoluble nickel compounds by inhalation during nickel refining correlated with increased incidences of lung and nasal cancers. Insoluble nickel subsulfide and nickel oxide (NiO) are carcinogenic in animals by
X Mao et al.
Cancer letters, 161(1), 57-62 (2000-11-18)
Differential display polymerase chain reaction (DD-PCR) was used to analyze the differentially expressed genes from nickel-transformed human embryonic lung (HEL) cells (MRC-9 and IMR-90) and their control counterparts (non-treated). Two genes, MS515 and IC82, were confirmed by Northern blot analysis.
Reactive oxygen-induced carcinogenesis causes hypermethylation of p16 (Ink4a) and activation of MAP kinase.
Govindarajan B, et al.
Molecular Medicine, 8(1) (2002)
M Okamoto
Differentiation; research in biological diversity, 61(5), 285-292 (1997-08-01)
It is well known that urodeles have the most powerful regenerative capacities among vertebrates, but there is little realisation that they are extremely resistant to spontaneous or chemically induced tumours. Regeneration and carcinogenesis have been considered to be two sides
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