推荐产品
方案
97%
表单
solid
mp
190-193 °C (lit.)
SMILES字符串
CCOc1ccc2nc(sc2c1)S(N)(=O)=O
InChI
1S/C9H10N2O3S2/c1-2-14-6-3-4-7-8(5-6)15-9(11-7)16(10,12)13/h3-5H,2H2,1H3,(H2,10,12,13)
InChI key
OUZWUKMCLIBBOG-UHFFFAOYSA-N
基因信息
human ... CA1(759) , CA12(771) , CA13(377677) , CA14(23632) , CA2(760) , CA3(761) , CA4(762) , CA5A(763) , CA5B(11238) , CA6(765) , CA7(766) , CA9(768)
mouse ... Car5a(12352)
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一般描述
6-乙氧基-2-苯并噻唑磺酰胺是一种膜渗透性碳酸酐酶抑制剂，已在正常白兔中对其眼部分布和药理学进行了研究。在体外引起抑制胰岛素降解 。
警示用语：
Warning
危险声明
危险分类
Eye Irrit. 2 - Skin Irrit. 2 - STOT SE 3
靶器官
Respiratory system
储存分类代码
11 - Combustible Solids
WGK
WGK 3
闪点(°F)
Not applicable
闪点(°C)
Not applicable
个人防护装备
dust mask type N95 (US), Eyeshields, Gloves
Holger M Becker et al.
The Journal of biological chemistry, 282(18), 13508-13521 (2007-03-14)
Several acid/base-coupled membrane transporters, such as the electrogenic sodium-bicarbonate cotransporter (NBCe1), have been shown to bind to different carbonic anhydrase isoforms to create a "transport metabolon." We have expressed NBCe1 derived from human kidney in oocytes of Xenopus leavis and
N A Tobey et al.
American journal of physiology. Gastrointestinal and liver physiology, 293(1), G264-G270 (2007-04-14)
The human esophagus is lined by stratified squamous epithelium (ESSE), and in some subjects with reflux disease the distal esophagus becomes lined by Barrett's specialized columnar epithelium (BSCE). ESSE and BSCE differ both histologically and functionally, the latter evident by
Jatinder Kaur et al.
Journal of medicinal chemistry, 63(17), 9540-9562 (2020-08-14)
Vancomycin-resistant enterococci (VRE) are the second leading cause of hospital-acquired infections (HAIs) attributed to a drug-resistant bacterium in the United States, and resistance to the frontline treatments is well documented. To combat VRE, we have repurposed the FDA-approved carbonic anhydrase
Studies of the effect of carbutamide on glucose-6-phosphatase.
C A KUETHER et al.
Diabetes, 6(1), 23-25 (1957-01-01)
Bernardo V Alvarez et al.
The Journal of physiology, 579(Pt 1), 127-145 (2006-11-25)
Hypertrophic cardiomyocyte growth contributes substantially to the progression of heart failure. Activation of the plasma membrane Na+-H+ exchanger (NHE1) and Cl- -HCO3- exchanger (AE3) has emerged as a central point in the hypertrophic cascade. Both NHE1 and AE3 bind carbonic
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