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蒸汽密度
3.46 (vs air)
蒸汽压
9.68 psi ( 20 °C)
方案
99%
表单
liquid
自燃温度
968 °F
包含
200 ppm MEHQ as inhibitor
expl. lim.
15.5 %
沸点
30-32 °C (lit.)
mp
−122 °C (lit.)
密度
1.213 g/mL at 20 °C (lit.)
储存温度
2-8°C
SMILES字符串
ClC(Cl)=C
InChI
1S/C2H2Cl2/c1-2(3)4/h1H2
InChI key
LGXVIGDEPROXKC-UHFFFAOYSA-N
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应用
1,1-二氯乙烯可作为单体，通过乳液聚合制备高分子乳胶复合材料。
警示用语：
Danger
危险分类
Acute Tox. 3 Oral - Acute Tox. 4 Inhalation - Carc. 2 - Eye Irrit. 2 - Flam. Liq. 1 - Skin Irrit. 2
储存分类代码
3 - Flammable liquids
WGK
WGK 3
闪点(°F)
-2.2 °F
闪点(°C)
-19 °C
个人防护装备
Eyeshields, Faceshields, Gloves
法规信息
危险化学品
Fan Chen et al.
Frontiers in microbiology, 9, 2306-2306 (2018-10-17)
Bioelectrochemical systems (BESs) are regarded as a promising approach for the enhanced dechlorination of chlorinated aliphatic hydrocarbons (CAHs). However, the electron distribution and transfer considering dechlorination, methanogenesis, and other bioprocesses in these systems are little understood. This study investigated the
1,1-Dichloroethylene.
Reviews of environmental contamination and toxicology, 106, 81-91 (1988-01-01)
Kimberly N Heck et al.
Journal of the American Chemical Society, 130(49), 16592-16600 (2009-06-26)
Insight into the nature of transient reaction intermediates and mechanistic pathways involved in heterogeneously catalyzed chemical reactions is obtainable from a number of surface spectroscopic techniques. Carrying out these investigations under actual reaction conditions is preferred but remains challenging, especially
Masarin Ban et al.
Toxicology, 184(1), 41-50 (2002-12-31)
Using immunotoxic functional tests, namely IgM response to sheep red blood cells (SRBCs) and interferon-gamma (IFN-gamma) production, this study simultaneously evaluated the effects of inhaled chloroform (10, 20, and 50 ppm), carbon tetrachloride (100, 200, and 300 ppm), 1,1-dichloroethylene (5
P G Forkert et al.
Drug metabolism and disposition: the biological fate of chemicals, 29(11), 1396-1402 (2001-10-17)
1,1-Dichloroethylene (DCE) causes hepatocellular necrosis that preferentially affects centrilobular hepatocytes. The cytotoxic lesion has been attributed to DCE oxidation mediated mainly by CYP2E1, resulting in formation of reactive intermediates including the DCE epoxide. Here, we have tested the hypothesis that
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