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Merck
CN

V900548

TWEEN® 20

Vetec™, reagent grade, 40%

Synonym(s):

Polyethylene glycol sorbitan monolaurate, Polyoxyethylenesorbitan monolaurate

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About This Item

CAS Number:
9005-64-5
UNSPSC Code:
12161900
MDL number:
MFCD00165986

Key Documents

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grade

reagent grade

description

non-ionic detergent

product line

Vetec™

assay

≥40%

form

viscous liquid

concentration

40%

technique(s)

DNA extraction: suitable, RNA extraction: suitable, cell culture | mammalian: suitable, protein purification: suitable

refractive index

n20/D 1.468 (lit.)

viscosity

400 mPa.s(25 °C)

mp

98.9 °C

solubility

water: 0.0002 g/L at 20 °C

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Application

Non-ionic detergent

Legal Information

TWEEN is a registered trademark of Croda International PLC
Vetec is a trademark of Merck KGaA, Darmstadt, Germany

Storage Class

10 - Combustible liquids

wgk

WGK 1

flash_point_f

>298.4 °F - closed cup

flash_point_c

> 148 °C - closed cup

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Certificates of Analysis (COA)

Lot/Batch Number
WXBF7028V
WXBF5069V
WXBF6153V
WXBF5949V
WXBF5137V

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Deng Pan et al.
Experimental brain research, 238(4), 1085-1095 (2020-03-28)
Ghrelin is an orexigenic agonist that acts directly on neurons in the hypothalamus, controlling appetite and energy balance. Although its role in appetite-associated neurons has been described, the relationship between peripheral ghrelin stimulation and oxytocin expression in the paraventricular nucleus
Julie Beegle et al.
Stem cells (Dayton, Ohio), 33(6), 1818-1828 (2015-02-24)
Mesenchymal stem cells/multipotent stromal cells (MSCs) are promising therapeutics for a variety of conditions. However, after transplantation, cell retention remains extremely challenging. Given that many hypoxic signals are transitory and that the therapeutic administration of MSCs is typically into tissues
Daniel C Zielinski et al.
Nature communications, 6, 7101-7101 (2015-06-10)
Drug side effects cause a significant clinical and economic burden. However, mechanisms of drug action underlying side effect pathogenesis remain largely unknown. Here, we integrate pharmacogenomic and clinical data with a human metabolic network and find that non-pharmacokinetic metabolic pathways

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