V000188
Petroleum ether
InChI key
ZRJNGFJIBZKXTP-UHFFFAOYSA-M
InChI
1S/C7H7.BrH.Mg/c1-7-5-3-2-4-6-7;;/h3-6H,1H3;1H;/q;;+1/p-1
vapor density
2.5 (vs air)
vapor pressure
25.8 psi ( 55 °C), 7.99 psi ( 20 °C)
autoignition temp.
475 °F
expl. lim.
8 %
refractive index
n20/D 1.363 (lit.)
density
0.64 g/mL at 25 °C (lit.)
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signalword
Danger
hcodes
Hazard Classifications
Aquatic Chronic 2 - Asp. Tox. 1 - Flam. Liq. 1 - Skin Irrit. 2 - STOT SE 3
target_organs
Central nervous system
Storage Class
3 - Flammable liquids
flash_point_f
-56.2 °F
flash_point_c
-49 °C
Regulatory Information
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Gunnar Damgård Nielsen et al.
Basic & clinical pharmacology & toxicology, 98(2), 115-123 (2006-02-01)
Numerous studies have suggested that long-term occupational exposure to white spirit may cause chronic toxic encephalopathy (WHO 1996). This review summarizes the chronic nervous system effects of white spirit in animal studies during a 30-year period. First, routine histopathology was
H R Lam et al.
Toxicology letters, 80(1-3), 39-48 (1995-10-01)
The effects of 3 weeks' or 6 months' inhalation exposure of rats to aromatic white spirit 6 h/day, 5 days/week at 0, 400, or 800 ppm were studied. Synaptosomal neurochemistry was investigated as index of the in situ conditions in
Lena Ernstgård et al.
Journal of applied toxicology : JAT, 29(3), 255-262 (2008-12-17)
A move from 'standard' white spirit (stdWS, 15-20% aromatics) to low-aromatic or dearomatized white spirit (deWS) has been seen, as the latter are considered to carry a smaller risk of health effects. However, data on health risks of deWS on
S Dauger et al.
Archives de pediatrie : organe officiel de la Societe francaise de pediatrie, 10(3), 221-223 (2003-06-28)
The Authors report a case of acute White-Spirit poisoning with pulmonary hypertension associated to respiratory distress syndrome. A 14-month-old infant drank an unknown quantity of White-Spirit while his parents were painting. After he spontaneously vomited, he presented a seizure at
Catherine F Poh et al.
Clinical cancer research : an official journal of the American Association for Cancer Research, 12(22), 6716-6722 (2006-11-24)
Genetically altered cells could become widespread across the epithelium of patients with oral cancer, often in clinically and histologically normal tissue, and contribute to recurrent disease. Molecular approaches have begun to yield information on cancer/risk fields; tissue optics could further
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