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Merck
CN

SAB2100476

Anti-CPT1A antibody produced in rabbit

affinity isolated antibody, lyophilized powder

Synonym(s):

Anti-CPT1, Anti-CPT1-L, Anti-Carnitine palmitoyltransferase 1A (liver), Anti-L-CPT1

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About This Item

UNSPSC Code:
12352203
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biological source

rabbit

conjugate

unconjugated

antibody form

affinity isolated antibody

antibody product type

primary antibodies

clone

polyclonal

form

lyophilized powder

mol wt

86 kDa

species reactivity

mouse, rat, pig, chicken, horse, rabbit, bovine, human, canine, zebrafish

technique(s)

western blot: suitable

immunogen sequence

LSTSQTPQQQVELFDLENNPEYVSSGGGFGPVADDGYGVSYILVGENLIN

UniProt accession no.

storage temp.

−20°C

Gene Information

human ... CPT1A(1374)

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Immunogen

Peptide region of the protein sequence according to NP_001027017.

Application

Applications in which this antibody has been used successfully, and the associated peer-reviewed papers, are given below.
Western Blotting (1 paper)

Physical form

Lyophilized from PBS buffer with 2% sucrose

Disclaimer

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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Storage Class

11 - Combustible Solids

wgk

WGK 2

flash_point_f

Not applicable

flash_point_c

Not applicable

Regulatory Information

常规特殊物品
低风险生物材料
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Laura Sanchez-Lazo et al.
Molecular endocrinology (Baltimore, Md.), 28(9), 1502-1521 (2014-07-25)
Oocyte meiotic maturation requires energy from various substrates including glucose, amino acids, and lipids. Mitochondrial fatty acid (FA) β-oxidation (FAO) in the oocyte is required for meiotic maturation, which is accompanied by differential expression of numerous genes involved in FAs
Zhaowei Cai et al.
BMC genomics, 16, 59-59 (2015-04-19)
Recent studies have indicated that low serum testosterone levels are associated with increased risk of developing hepatic steatosis; however, the mechanisms mediating this phenomenon have not been fully elucidated. To gain insight into the role of testosterone in modulating hepatic

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