S2378
Sodium succinate dibasic hexahydrate
ReagentPlus®, ≥99%
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All Photos(4)
Synonym(s):
Butanedioic acid disodium salt, Succinic acid disodium salt
Linear Formula:
NaOOCCH2CH2COONa · 6H2O
CAS Number:
Molecular Weight:
270.14
Beilstein:
3921044
EC Number:
MDL number:
UNSPSC Code:
12352100
PubChem Substance ID:
NACRES:
NA.21
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Quality Level
product line
ReagentPlus®
Assay
≥99%
solubility
H2O: 100 mg/mL, clear, colorless
SMILES string
O.O.O.O.O.O.[Na+].[Na+].[O-]C(=O)CCC([O-])=O
InChI
1S/C4H6O4.2Na.6H2O/c5-3(6)1-2-4(7)8;;;;;;;;/h1-2H2,(H,5,6)(H,7,8);;;6*1H2/q;2*+1;;;;;;/p-2
InChI key
ZBTUYCUNQBRXOR-UHFFFAOYSA-L
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Application
Sodium succinate dibasic hexahydrate can be used as an organic additive in the preparation of well-defined hollow nano-baskets morphology of ZnO nanoparticles (ZnO-HNBs) via a low-temperature solution method using zinc nitrate hexahydrate and hexamethylenetetramine. ZnO-HNBs are further applied as photocatalysts for dye degradation.
Legal Information
ReagentPlus is a registered trademark of Merck KGaA, Darmstadt, Germany
WGK
WGK 1
Flash Point(F)
Not applicable
Flash Point(C)
Not applicable
Personal Protective Equipment
dust mask type N95 (US), Eyeshields, Gloves
Certificates of Analysis (COA)
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Jin Seop Ahn et al.
International journal of molecular sciences, 19(10) (2018-10-03)
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Oswaldo A Lozoya et al.
Cell reports, 32(11), 108131-108131 (2020-09-17)
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Jean-Philippe Leduc-Gaudet et al.
The Journal of physiology, 597(7), 1975-1991 (2019-01-08)
Recent evidence suggests that impaired mitophagy, a process in charge of removing damaged/dysfunctional mitochondria and in part regulated by Parkin, could contribute to the ageing-related loss of muscle mass and function. In the present study, we show that Parkin overexpression
Thamir Alandijany et al.
Journal of neurovirology, 19(6), 537-549 (2013-11-28)
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Marta Murgia et al.
Cell reports, 29(12), 3825-3834 (2019-12-19)
The mosaic distribution of cytochrome c oxidase+ (COX+) and COX- muscle fibers in mitochondrial disorders allows the sampling of fibers with compensated and decompensated mitochondrial function from the same individual. We apply laser capture microdissection to excise individual COX+ and
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