310166
Sodium chloride
reagent grade, ≥98%, +80 mesh particle size
Synonym(s):
Halite, NaCl
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About This Item
Linear Formula:
NaCl
CAS Number:
Molecular Weight:
58.44
Beilstein:
3534976
EC Number:
MDL number:
UNSPSC Code:
12352302
PubChem Substance ID:
Assay:
≥98%
grade:
reagent grade
form:
powder
solubility:
water: soluble (358 g/l at 20 °C)
Recommended Products
grade
reagent grade
vapor pressure
1 mmHg ( 865 °C)
Assay
≥98%
form
powder
particle size
+80 mesh
mp
801 °C (lit.)
solubility
water: soluble (358 g/l at 20 °C)
SMILES string
[Na+].[Cl-]
InChI
1S/ClH.Na/h1H;/q;+1/p-1
InChI key
FAPWRFPIFSIZLT-UHFFFAOYSA-M
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Other Notes
This product has been replaced by S9625-Sigma-Aldrich | Sodium chloride, ReagentPlus®, ≥99.5% (titration) | NaCl
Legal Information
ReagentPlus is a registered trademark of Merck KGaA, Darmstadt, Germany
Storage Class Code
13 - Non Combustible Solids
WGK
WGK 1
Flash Point(F)
Not applicable
Flash Point(C)
Not applicable
Personal Protective Equipment
dust mask type N95 (US), Eyeshields, Gloves
Regulatory Information
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Matthew Gallon et al.
Proceedings of the National Academy of Sciences of the United States of America, 111(35), E3604-E3613 (2014-08-20)
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Zlatko Kopecki et al.
Oncotarget, 6(34), 36426-36440 (2015-10-27)
Flightless I (Flii) is an actin remodeling protein that affects cellular processes including adhesion, proliferation and migration. In order to determine the role of Flii during carcinogenesis, squamous cell carcinomas (SCCs) were induced in Flii heterozygous (Flii+/-), wild-type and Flii
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Diabetologia, 57(8), 1635-1644 (2014-05-29)
Hypoxic damage complicates islet isolation for transplantation and may contribute to beta cell failure in type 2 diabetes. Polymorphisms in the SLC30A8 gene, encoding the secretory granule zinc transporter 8 (ZnT8), influence type 2 diabetes risk, conceivably by modulating cytosolic
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Proceedings of the National Academy of Sciences of the United States of America, 111(28), E2841-E2850 (2014-07-02)
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Cancer research, 75(23), 5058-5069 (2015-11-19)
Elevated oxidative stress is an aberration seen in many solid tumors, and exploiting this biochemical difference has the potential to enhance the efficacy of anticancer agents. Homeostasis of reactive oxygen species (ROS) is important for normal cell function, but excessive
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