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Merck
CN

09180

3-Amino-4-hydroxybenzenesulfonic acid

technical, ≥95% (T)

Synonym(s):

2-Aminophenol-4-sulfonic acid

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About This Item

Linear Formula:
H2NC6H3(OH)SO3H
CAS Number:
Molecular Weight:
189.19
UNSPSC Code:
12352100
NACRES:
NA.22
PubChem Substance ID:
EC Number:
202-662-8
Beilstein/REAXYS Number:
514765
MDL number:
Assay:
≥95% (T)
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InChI key

ULUIMLJNTCECJU-UHFFFAOYSA-N

InChI

1S/C6H7NO4S/c7-5-3-4(12(9,10)11)1-2-6(5)8/h1-3,8H,7H2,(H,9,10,11)

SMILES string

Nc1cc(ccc1O)S(O)(=O)=O

grade

technical

assay

≥95% (T)

ign. residue

≤2%

mp

≥300 °C (lit.)

functional group

sulfonic acid

Quality Level

pictograms

Exclamation mark

signalword

Warning

hcodes

Hazard Classifications

Eye Irrit. 2 - Skin Irrit. 2

Storage Class

8A - Combustible corrosive hazardous materials

wgk

WGK 1

ppe

Eyeshields, Faceshields, Gloves, type P3 (EN 143) respirator cartridges


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[Abstracts of the 116th Internal Medicine Congress. April 10-14, 2010, Wiesbaden, Germany].
Medizinische Klinik (Munich, Germany : 1983), 105 Suppl 1, 6-113 (2010-04-14)
Milena M Cojic et al.
Oxidative medicine and cellular longevity, 2021, 7942716-7942716 (2021-07-10)
Recent advances in vitamin D research indicate that patients with type 2 diabetes mellitus (T2DM) are suffering from vitamin D deficiency and increased oxidative stress to a variable extent, which could produce different health impacts for each individual. The novel
Nicole Kemberly R Rocha et al.
Frontiers in neuroscience, 14, 579984-579984 (2020-12-19)
Neuronal-glial interactions are critical for brain homeostasis, and disruption of this process may lead to excessive glial activation and inadequate pro-inflammatory responses. Abnormalities in neuronal-glial interactions have been reported in the pathophysiology of Alzheimer's disease (AD), where lithium has been
Edjair V Cabral et al.
PloS one, 7(8), e43791-e43791 (2012-08-29)
High Na(+) intake is a reality in nowadays and is frequently accompanied by renal and cardiovascular alterations. In this study, renal mechanisms underlying perinatal Na(+) overload-programmed alterations in Na(+) transporters and the renin/angiotensin system (RAS) were investigated, together with effects
Cyrus Daneshvar et al.
Clinical infectious diseases : an official publication of the Infectious Diseases Society of America, 49(6), 852-860 (2009-07-29)
Plasmodium knowlesi is increasingly recognized as a cause of human malaria in Southeast Asia but there are no detailed prospective clinical studies of naturally acquired infections. In a systematic study of the presentation and course of patients with acute P.

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