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SLHV004SL

Millipore

Millex® PVDF syringe filter

pore size 0.45 μm, diam. 4 mm, sterile, hydrophilic

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Synonym(s):
0.45 μm PVDF syringe filter, Durapore® PVDF syringe filter, Millex-HV 0.45µm Filter Unit, Millex-HV Filter Unit, Millex-HV syringe filter, disposable syringe filter, sterile syringe filter, syringe filter
UNSPSC Code:
41104922
eCl@ss:
32031690
NACRES:
NB.22

material

PVDF membrane
high-density polyethylene housing

Quality Level

sterility

ethylene oxide treated
sterile

product line

Millex®

feature

holdup volume< 10 μL
hydrophilic

manufacturer/tradename

Millex®

parameter

14 bar max. inlet pressure (200 psi)
45 °C max. temp.

technique(s)

low protein-binding filtration: suitable
sterile filtration: suitable

diam.

4 mm

filtration area

0.1 cm2

housing diam.

6.4 mm

inlet to outlet H

19.7 mm

volume

1 mL

matrix

Durapore®

pore size

0.45 μm

input

aqueous solution(s)
tissue culture medium
mild organic solvent(s)

fitting

female Luer-Lok® inlet
male Luer outlet stepped slip

shipped in

ambient

General description

“Millex” PVDF syringe filter utilizes PVDF membrane of pore size 0.45 μm and high-density polyethylene housing.

Application

“Millex” PVDF syringe filter is intended for sterile filtration of tissue culture media, additives, buffers, biological solutions.

Features and Benefits

Designed to ensure lowest binding for protein rich solutions.

Legal Information

Durapore is a registered trademark of Merck KGaA, Darmstadt, Germany
Luer-Lok is a registered trademark of Becton-Dickinson & Co.
Millex is a registered trademark of Merck KGaA, Darmstadt, Germany

Certificates of Analysis (COA)

Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.

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Removal of residual colonic ciprofloxacin in the rat by activated charcoal entrapped within zinc-pectinate beads.
Khoder, Mouhamad, et al.
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Pancreatic ductal adenocarcinoma (PDAC) can be divided into transcriptomic subtypes with two broad lineages referred to as classical (pancreatic) and squamous. We find that these two subtypes are driven by distinct metabolic phenotypes. Loss of genes that drive endodermal lineage

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