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Merck
CN

S-024

HU-210 solution

100 μg/mL in methanol, ampule of 1 mL, (Spice Cannabinoid), certified reference material, Cerilliant®

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About This Item

Empirical Formula (Hill Notation):
C25H38O3
CAS Number:
112830-95-2
Molecular Weight:
386.57
NACRES:
NA.24
UNSPSC Code:
41116107
EC Number:
200-659-6

Key Documents

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InChI key

SSQJFGMEZBFMNV-WOJBJXKFSA-N

InChI

1S/C25H38O3/c1-6-7-8-9-12-24(2,3)18-14-21(27)23-19-13-17(16-26)10-11-20(19)25(4,5)28-22(23)15-18/h10,14-15,19-20,26-27H,6-9,11-13,16H2,1-5H3/t19-,20-/m1/s1

SMILES string

CCCCCCC(C)(C)c1cc(O)c2[C@@H]3CC(CO)=CC[C@H]3C(C)(C)Oc2c1

grade

certified reference material

Quality Level

300

feature

Snap-N-Spike®/Snap-N-Shoot®

packaging

ampule of 1 mL

manufacturer/tradename

Cerilliant®

drug control

Narcotic Licence Schedule E (Switzerland)

concentration

100 μg/mL in methanol

technique(s)

gas chromatography (GC): suitable, liquid chromatography (LC): suitable

format

single component solution

storage temp.

−20°C

Related Categories

General description

HU-210 is a synthetic cannabinoid found in the herbal mixture “Spice.” Illicit use of this Spice cannabinoid includes smoking the herbal product to obtain a psychoactive effect. Structurally similar to (-)-Δ8-THC and 11-Hydroxy-Δ9-THC, HU-210 is considerably more potent than natural THC from cannabis and has an extended duration of action.
In July, 2009, the US Drug Enforcement Agency (DEA) placed HU-210 on its Drugs and Chemicals of Concern list. Since HU-210′s placement on this list, DEA has reported seizures of Spice herbal mixtures in Ohio and Florida. HU-210 is currently listed as a Schedule I controlled substance. In recognition of this regulatory challenge, Cerilliant supplies its certified HU-210 reference standard in a convenient, quantitative, DEA-exempt solution.

Legal Information

CERILLIANT is a registered trademark of Merck KGaA, Darmstadt, Germany
Snap-N-Shoot is a registered trademark of Cerilliant Corporation
Snap-N-Spike is a registered trademark of Merck KGaA, Darmstadt, Germany

signalword

Danger

Hazard Classifications

Acute Tox. 3 Dermal - Acute Tox. 3 Inhalation - Acute Tox. 3 Oral - Flam. Liq. 2 - STOT SE 1

target_organs

Eyes

Storage Class

3 - Flammable liquids

wgk

WGK 1

flash_point_f

49.5 °F - closed cup

flash_point_c

9.7 °C - closed cup

Regulatory Information

监管及禁止进口产品
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Lidia Martín-Couce et al.
Angewandte Chemie (International ed. in English), 51(28), 6896-6899 (2012-06-13)
Receptors made visible: The described biotin-tagged small-molecule probes with excellent affinities for the CB(1) and CB(2) cannabinoid receptors (CB(1)R and CB(2)R) enable direct visualization of these receptors in native cellular systems, including neurons, microglia, and immune cells. This method could
C L Limebeer et al.
British journal of pharmacology, 167(5), 1126-1136 (2012-06-08)
Conditioned gaping reactions reflect nausea-induced behaviour in rats. Cannabinoid 1 receptor (CB(1) ) agonists interfere with the establishment of nausea-induced conditioned gaping; however, it is not known if their effects are mediated by an action at peripheral or central CB(1)
Vu H Nguyen et al.
Pharmacology, 89(1-2), 64-73 (2012-02-04)
Functional linkages between the cannabinoid CB(1) and the dopaminergic systems have been reported although the observations and the mechanisms hypothesizing their interactions at the G protein-coupled receptor (GPCR) functionality level are conflicting. Administration of a potent cannabinoid agonist, HU210, at
Vu H Nguyen et al.
Brain research bulletin, 87(2-3), 172-179 (2011-12-14)
Cannabis use has been shown to alter brain metabolism in both rat models and humans although the observations between both species are conflicting. In the present study, we examined the short term effects of a single-dose injection of the synthetic
Ming-hua Cao et al.
PloS one, 7(12), e52921-e52921 (2013-01-04)
Acute pancreatitis (AP), especially severe acute pancreatitis often causes extra-pancreatic complications, such as acute gastrointestinal mucosal lesion (AGML) which is accompanied by a considerably high mortality, yet the pathogenesis of AP-induced AGML is still not fully understood. In this report
View All Related Papers

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