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Merck
CN

E-041

Ethanol-150

150 mg/dL in H2O, ampule of 10 × 1.2 mL, certified reference material, Cerilliant®

Synonym(s):

Ethanol solution, Ethyl alcohol

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About This Item

Linear Formula:
C2H5OH
CAS Number:
64-17-5
Molecular Weight:
46.07
UNSPSC Code:
41116107
NACRES:
NA.24
PubChem Substance ID:
329798969
MDL number:
MFCD00003568
Beilstein/REAXYS Number:
1718733

Key Documents

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InChI

1S/C2H6O/c1-2-3/h3H,2H2,1H3

SMILES string

CCO

InChI key

LFQSCWFLJHTTHZ-UHFFFAOYSA-N

grade

certified reference material

form

liquid

feature

Snap-N-Spike®/Snap-N-Shoot®

packaging

ampule of 10 × 1.2 mL

manufacturer/tradename

Cerilliant®

concentration

150 mg/dL in H2O

technique(s)

gas chromatography (GC): suitable, liquid chromatography (LC): suitable

application(s)

forensics and toxicology

format

single component solution

storage temp.

2-8°C

Quality Level

300

Related Categories

General description

A kit of NIST traceable ethanol standards suitable for calibration of ethanol testing methods. These Certified Reference Materials are widely used in forensic analysis and clinical toxicology applications for determination of blood alcohol content.

Legal Information

CERILLIANT is a registered trademark of Merck KGaA, Darmstadt, Germany
Snap-N-Shoot is a registered trademark of Cerilliant Corporation
Snap-N-Spike is a registered trademark of Merck KGaA, Darmstadt, Germany

Storage Class

10 - Combustible liquids

wgk

nwg

flash_point_f

Not applicable

flash_point_c

Not applicable

Certificates of Analysis (COA)

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S Enocksson et al.
The Journal of clinical investigation, 95(5), 2239-2245 (1995-05-01)
Although it is well established in several mammalian species that beta 3-adrenoceptors play a major role in regulating lipolysis and thermogenesis in adipose tissue, the functional existence and role of this receptor subtype in man has been controversial. We investigated
Kazue Hashimoto-Torii et al.
Neuron, 82(3), 560-572 (2014-04-15)
Prenatal exposure of the developing brain to various environmental challenges increases susceptibility to late onset of neuropsychiatric dysfunction; still, the underlying mechanisms remain obscure. Here we show that exposure of embryos to a variety of environmental factors such as alcohol
Irene Zelner et al.
Drug metabolism reviews, 45(3), 277-299 (2013-05-30)
The ability to undergo non-oxidative metabolism from ethanol to fatty acid ethyl esters (FAEEs) varies greatly among tissues and organs. To gain a greater understanding of non-oxidative ethanol metabolism to FAEE, we aimed to collect all published data on FAEE
Elhaseen Elamin et al.
American journal of physiology. Gastrointestinal and liver physiology, 306(8), G677-G685 (2014-02-22)
Evidence indicates that ethanol-induced intestinal barrier dysfunction and subsequent endotoxemia plays a key role in the pathogenesis of alcoholic liver disease. Recently, it has been demonstrated that ethanol induces RhoA kinase activation in intestinal epithelium, thereby disrupting barrier integrity. In
J Piskur et al.
FEBS letters, 375(3), 174-178 (1995-11-20)
A recently developed mechanochemical method has provided a new, efficient tool for studies on the thermal stability and structure of aggregated DNA in ethanol-water solutions. At low ethanol concentrations DNA is fully soluble and is in the B form. However
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