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About This Item
Empirical Formula (Hill Notation):
C8H5F3O2S
CAS Number:
Molecular Weight:
222.18
UNSPSC Code:
12352100
NACRES:
NA.22
PubChem Substance ID:
EC Number:
206-316-7
Beilstein/REAXYS Number:
168645
MDL number:
Product Name
2-Thenoyltrifluoroacetone, 99%
InChI key
TXBBUSUXYMIVOS-UHFFFAOYSA-N
InChI
1S/C8H5F3O2S/c9-8(10,11)7(13)4-5(12)6-2-1-3-14-6/h1-3H,4H2
SMILES string
FC(F)(F)C(=O)CC(=O)c1cccs1
assay
99%
bp
96-98 °C/8 mmHg (lit.)
mp
40-44 °C (lit.)
storage temp.
2-8°C
Quality Level
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Related Categories
Application
Reagent for the determination of actinides and lanthanides.
Storage Class
11 - Combustible Solids
wgk
WGK 3
flash_point_f
233.6 °F - closed cup
flash_point_c
112 °C - closed cup
ppe
dust mask type N95 (US), Eyeshields, Gloves
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Jeffery, G.H. et al.
Vogel's Textbook of Quantitative Chemical Analysis, 170-170 (1989)
Vladimir A Kokorekin et al.
Molecules (Basel, Switzerland), 25(18) (2020-09-17)
In this article, we demonstrate how an original effective "metal-free" and "chromatography-free" route for the synthesis of 3-thiocyanatopyrazolo[1,5-a]pyrimidines has been developed. It is based on electrooxidative (anodic) C-H thiocyanation of 5-aminopyrazoles by thiocyanate ion leading to 4-thiocyanato-5-aminopyrazoles (stage 1, yields
Yongqiang Chen et al.
Journal of cell science, 120(Pt 23), 4155-4166 (2007-11-23)
Autophagy is a self-digestion process important for cell survival during starvation. It has also been described as a form of programmed cell death. Mitochondria are important regulators of autophagy-induced cell death and damaged mitochondria are often degraded by autophagosomes. Inhibition
Yajuan Li et al.
Dalton transactions (Cambridge, England : 2003), 39(10), 2554-2562 (2010-02-25)
A series of novel ternary organic-inorganic mesoporous polymeric hybrids TTFA-S16-Eu-PMMA, TTFA-S16-Eu-PMAA, and TTFA-S16-Eu-PVP (TTFA = 2-Thenoyltrifluoroacetone; PMMA = polymethyl methacrylate; PMAA = polymethacrylic acid; PVP = polyvinylpyrrolidone) have been assembled by the Eu(3+) complex covalently attaching to the TTFA directly
Stefan Dröse et al.
Molecular pharmacology, 79(5), 814-822 (2011-02-01)
In this study, we have analyzed the effect of different cardioprotective complex II inhibitors on the mitochondrial production of reactive oxygen species (ROS) because ROS seem to be essential for signaling during preconditioning to prevent ischemia/reperfusion injury. Despite different binding
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