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Safety Information

HT-CDOAC100

Cadmium(II) acetate

SAFC Hitech®, anhydrous, 99.995%

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Linear Formula:
Cd(OCOCH3)2
CAS Number:
543-90-8
Molecular Weight:
230.50
MDL number:
MFCD00012449
UNSPSC Code:
12352200
PubChem Substance ID:
329815278

grade

anhydrous

product line

SAFC Hitech®

Assay

99.995%

mp

255-273 °C

SMILES string

CC(=O)O[Cd]OC(C)=O

InChI

1S/2C2H4O2.Cd/c2*1-2(3)4;/h2*1H3,(H,3,4);/q;;+2/p-2

InChI key

LHQLJMJLROMYRN-UHFFFAOYSA-L

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Legal Information

SAFC Hitech is a registered trademark of Sigma-Aldrich Co. LLC

Signal Word

Danger

Hazard Classifications

Acute Tox. 4 Dermal - Acute Tox. 4 Inhalation - Acute Tox. 4 Oral - Aquatic Acute 1 - Aquatic Chronic 1 - Carc. 1B - Muta. 1B - STOT RE 1 Oral

Storage Class Code

6.1C - Combustible, acute toxic Cat.3 / toxic compounds or compounds which causing chronic effects

WGK

WGK 3

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Regulatory Information

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R Saravanan et al.
Journal of colloid and interface science, 452, 126-133 (2015-05-04)
A ternary ZnO/Ag/CdO nanocomposite was synthesized using thermal decomposition method. The resulting nanocomposite was characterized by X-ray diffraction, field emission scanning electron microscopy, transmission electron microscopy, UV-Vis spectroscopy, and X-ray photoelectron spectroscopy. The ZnO/Ag/CdO nanocomposite exhibited enhanced photocatalytic activity under
M Lag et al.
Cell biology and toxicology, 18(1), 29-42 (2002-05-07)
The lung is a target organ for cadmium (Cd) toxicity. Apoptosis induced by cadmium acetate (CdAc) was studied in alveolar type 2 cells and Clara cells isolated from rat lung. Relatively low concentrations of CdAc (1-10 micromol/L) induced apoptosis after
L M Cavaco et al.
Antimicrobial agents and chemotherapy, 54(9), 3605-3608 (2010-06-30)
We recently reported a phenotypic association between reduced susceptibility to zinc and methicillin resistance in Staphylococcus aureus CC398 isolates from Danish swine (F. M. Aarestrup, L. M. Cavaco, and H. Hasman, Vet. Microbiol. 142:455-457, 2009). The aim of this study
Marit Låg et al.
Toxicology, 211(3), 253-264 (2005-06-01)
Cadmium acetate (CdAc) induced apoptosis in primary alveolar type 2 cells and Clara cells from rat lung. Phosphorylation of the MAPKs ERK1/2, p38 and JNK was markedly increased in both cell types 15 min to 2 h after start of
J I Chao et al.
Mutation research, 498(1-2), 7-18 (2001-10-24)
Previously, we have demonstrated that cadmium acetate significantly induces hprt mutation frequency in Chinese hamster ovary (CHO)-K1 and that 3-amino-1,2,4-triazole (3AT), a catalase inhibitor, potentiates the mutagenicity of cadmium [Chem. Res. Toxicol. 9 (1996) 1360-1367]. In this study, we investigate
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