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Merck
CN

A21207

3-Acetylpyridine

98%

Synonym(s):

Methyl 3-pyridyl ketone

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About This Item

Empirical Formula (Hill Notation):
C7H7NO
CAS Number:
Molecular Weight:
121.14
UNSPSC Code:
12352100
NACRES:
NA.22
PubChem Substance ID:
EC Number:
206-496-7
Beilstein/REAXYS Number:
107751
MDL number:
Assay:
98%
Form:
liquid
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InChI

1S/C7H7NO/c1-6(9)7-3-2-4-8-5-7/h2-5H,1H3

InChI key

WEGYGNROSJDEIW-UHFFFAOYSA-N

SMILES string

CC(=O)c1cccnc1

assay

98%

form

liquid

Quality Level

bp

220 °C (lit.)

mp

11-13 °C (lit.)

density

1.102 g/mL at 25 °C (lit.)

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Danger

hcodes

Hazard Classifications

Acute Tox. 3 Oral - Skin Irrit. 2

Storage Class

6.1C - Combustible acute toxic Cat.3 / toxic compounds or compounds which causing chronic effects

wgk

WGK 1

flash_point_f

219.2 °F - closed cup

flash_point_c

104 °C - closed cup

ppe

Eyeshields, Faceshields, Gloves, type ABEK (EN14387) respirator filter


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Matasha Dhar et al.
Nature communications, 7, 12938-12938 (2016-09-22)
Neurodegenerative lesions induce sprouting of new collaterals from surviving axons, but the extent to which this form of axonal remodelling alters brain functional structure remains unclear. To understand how collateral sprouting proceeds in the adult brain, we imaged post-lesion sprouting
Huawen Lin et al.
PLoS genetics, 6(9), e1001105-e1001105 (2010-09-15)
The essential coenzyme nicotinamide adenine dinucleotide (NAD+) plays important roles in metabolic reactions and cell regulation in all organisms. Bacteria, fungi, plants, and animals use different pathways to synthesize NAD+. Our molecular and genetic data demonstrate that in the unicellular
Iran Goudarzi et al.
European journal of pharmacology, 642(1-3), 56-65 (2010-07-20)
Electrophysiological dysfunction of Purkinje cells causes cerebellar ataxia. Recent studies indicated that 4-aminopyridine (4-AP) can prevent the attacks in patients with episodic ataxia type 2. However, the cellular mechanism(s) by which 4-AP might be beneficial for the improvement of motor
Ai-Li Cai et al.
The European journal of neuroscience, 24(8), 2169-2176 (2006-10-18)
Zinc neurotoxicity has been demonstrated in ischemic, seizure, hypoglycemic, and trauma-induced neuronal death where Zn(2+) is thought to be synaptically released and taken up in neighbouring neurons, reaching toxic concentrations. We previously demonstrated that toxicity of extracellular Zn(2+) depended on
Robin J Harvey et al.
The European journal of neuroscience, 21(7), 1777-1784 (2005-05-05)
Previous experiments have shown that in the mature cerebellum both blocking of spontaneous electrical activity and destruction of the climbing fibres by a lesion of the inferior olive have a similar profound effect on the spine distribution on the proximal

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