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Merck
CN
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B5774

Supelco

苯溴马隆

analytical standard

别名:

(3,5-二溴-4-羟基苯基)(2-乙基-3-苯并呋喃基)甲酮

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100 TESTS
CN¥13,220.39

CN¥13,220.39

预计发货时间August 05, 2025详情

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100 TESTS
CN¥13,220.39

About This Item

经验公式（希尔记法）:
C17H12Br2O3
CAS号:
3562-84-3
分子量:
424.08
EC 号:
222-630-7
MDL编号:
MFCD00078962
UNSPSC代码:
41116107
PubChem化学物质编号:
329773266
NACRES:
NA.24

CN¥13,220.39

预计发货时间August 05, 2025详情

获取大包装报价

等级

analytical standard

质量水平

200

方案

~95%

技术

HPLC: suitable
gas chromatography (GC): suitable

应用

forensics and toxicology
pharmaceutical (small molecule)
veterinary

包装形式

neat

储存温度

2-8°C

SMILES字符串

CCc1oc2ccccc2c1C(=O)c3cc(Br)c(O)c(Br)c3

InChI

1S/C17H12Br2O3/c1-2-13-15(10-5-3-4-6-14(10)22-13)16(20)9-7-11(18)17(21)12(19)8-9/h3-8,21H,2H2,1H3

InChI key

WHQCHUCQKNIQEC-UHFFFAOYSA-N

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71015BC

GDS MPX Top 7 STEC

manufacturer/tradename

Assurance GDS®, BioControl Systems 71018-100

manufacturer/tradename

Assurance GDS®, BioControl Systems 71017-100

manufacturer/tradename

Assurance GDS®, BioControl Systems 71005-100

manufacturer/tradename

Assurance GDS®, BioControl Systems 71015-100, BioControl Systems 71015-576

suitability

enterovirulent E. coli (EEC) (shiga toxigenic)

suitability

enterovirulent E. coli (EEC) (shiga toxigenic)

suitability

Escherichia coli (O157:H7)

suitability

enterovirulent E. coli (EEC) (shiga toxigenic)

compatibility

for use with GDS Rotor-Gene

compatibility

for use with GDS Rotor-Gene

compatibility

for use with GDS Rotor-Gene

compatibility

for use with GDS Rotor-Gene

Quality Level

200

Quality Level

200

Quality Level

200

Quality Level

200

technique(s)

PCR: suitable

technique(s)

PCR: suitable

technique(s)

PCR: suitable

technique(s)

PCR: suitable

application(s)

food and beverages
pathogen testing

application(s)

food and beverages
pathogen testing

application(s)

food and beverages
pathogen testing

application(s)

food and beverages
pathogen testing

应用

有关合适仪器技术的更多信息，请参考产品′分析证书。如需进一步支持，请联系技术服务。

象形图

Skull and crossbones
GHS06

警示用语：

Danger

危险声明

H301

预防措施声明

P301 + P310

危险分类

Acute Tox. 3 Oral

储存分类代码

6.1C - Combustible acute toxic Cat.3 / toxic compounds or compounds which causing chronic effects

WGK

WGK 3

闪点(°F)

Not applicable

闪点(°C)

Not applicable

个人防护装备

dust mask type N95 (US), Eyeshields, Faceshields, Gloves

    • 历史批次信息供参考:

    分析证书(COA)

    Lot/Batch Number
    0000432308
    0000432308
    0000405122
    0000405122
    0000398407

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    如果您需要特殊版本，可通过批号或批次号查找具体证书。

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    在文件库中查找您最近购买产品的文档。

    访问文档库

    Zhijian Lin et al.
    Journal of medicinal food, 17(11), 1214-1221 (2014-10-15)
    Inulin, a group of dietary fibers, is reported to improve the metabolic disorders. In the present study, we investigated the effects of chicory inulin on serum metabolites of uric acid (UA), lipids, glucose, and abdominal fat deposition in quail model
    Ming-Han H Lee et al.
    Drug safety, 31(8), 643-665 (2008-07-19)
    Benzbromarone, a potent uricosuric drug, was introduced in the 1970s and was viewed as having few associated serious adverse reactions. It was registered in about 20 countries throughout Asia, South America and Europe. In 2003, the drug was withdrawn by
    Kaoru Kobayashi et al.
    Biopharmaceutics & drug disposition, 33(8), 466-473 (2012-08-31)
    Benzbromarone (BBR) is metabolized to 1'-hydroxy BBR and 6-hydroxy BBR in the liver. 6-Hydroxy BBR is further metabolized to 5,6-dihydroxy BBR. The aim of this study was to identify the CYP isozymes involved in the metabolism of BBR to 1'-hydroxy
    Tip W Loo et al.
    Biochemistry, 50(21), 4393-4395 (2011-04-28)
    Deletion of Phe508 from the first nucleotide-binding domain of the CFTR chloride channel causes cystic fibrosis because it inhibits protein folding. Indirect approaches such as incubation at low temperatures can partially rescue ΔF508 CFTR, but the protein is unstable at
    Fernanda Cristina Mazali et al.
    Nephron. Experimental nephrology, 120(1), e12-e19 (2011-12-01)
    Hyperuricemia frequently complicates cyclosporine (CsA) therapy. Previous studies have shown that hyperuricemia exacerbates interstitial and vascular lesions in the cyclosporine model. We tested the hypothesis that normalization of uric acid could prevent the development of cyclosporine toxicity. CsA nephropathy was
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