推荐产品
生物来源
rat
质量水平
抗体形式
culture supernatant
抗体产品类型
primary antibodies
克隆
NC1, monoclonal
种属反应性
human
种属反应性（根据同源性预测）
mammals
制造商/商品名称
Chemicon®
技术
immunohistochemistry: suitable
同位素/亚型
IgG2a
UniProt登记号
运输
dry ice
靶向翻译后修饰
unmodified
基因信息
human ... TAC1(6863)
特异性
与P物质的COOH末端反应。与Leu-或Met-脑啡肽、生长抑素或β-内啡肽无交叉反应。5%依来多新。
免疫原
与BSA偶联的P物质。
应用
以1:100 - 1:200对人脑进行免疫组织化学。
最佳工作稀释度必须由最终使用者进行确定。
最佳工作稀释度必须由最终使用者进行确定。
使用经验证可用于IH的抗P物质抗体（pain，克隆NC1）检测P物质。
外形
冻干上清液，不含防腐剂。
储存及稳定性
冻干抗体可在2-8°C下保存长达3个月，长期保存应在-20°C下保存。用100 μL无菌DI水复溶。将未稀释的等分试样在-20°C下保存长达6个月。应避免反复冻/融循环。
其他说明
浓度：关于批次特定浓度请参见检验报告。
法律信息
CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany
警示用语：
Warning
危险声明
危险分类
Eye Irrit. 2 - Skin Irrit. 2
WGK
WGK 2
High-fat diet: bacteria interactions promote intestinal inflammation which precedes and correlates with obesity and insulin resistance in mouse.
Ding, S; Chi, MM; Scull, BP; Rigby, R; Schwerbrock, NM; Magness, S; Jobin, C; Lund, PK
Testing null
Liang Han et al.
Nature neuroscience, 21(3), 324-328 (2018-02-07)
Asthma, accompanied by lung inflammation, bronchoconstriction and airway hyper-responsiveness, is a significant public health burden. Here we report that Mas-related G protein-coupled receptors (Mrgprs) are expressed in a subset of vagal sensory neurons innervating the airway and mediates cholinergic bronchoconstriction
Hypoalgesia in mice with a targeted deletion of the tachykinin 1 gene.
Zimmer, A; Zimmer, AM; Baffi, J; Usdin, T; Reynolds, K; Konig, M; Palkovits, M; Mezey, E
Proceedings of the National Academy of Sciences of the USA null
Tibor Z Veres et al.
American journal of respiratory cell and molecular biology, 37(5), 553-561 (2007-06-30)
Neuroimmune interactions play a critical role in the pathogenesis of asthma. Symptoms like wheezing and cough have been attributed to neural dysregulation, whereas sensitization and the induction of allergic inflammation have been linked with the activity of dendritic cells. Neuropeptides
Andrew P Ray et al.
Pharmacology, biochemistry, and behavior, 94(1), 211-218 (2009-08-25)
Research on the mechanisms of emesis has implicated multiple neurotransmitters via both central (dorsal vagal complex) and peripheral (enteric neurons and enterochromaffin cells) anatomical substrates. Taking advantage of advances in receptor-specific agonists, and utilizing Fos expression as a functional activity
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